Krystal Angeline Hay
Physiatrist (Physical Medicine) | Physical Medicine & Rehabilitation
301 Wolverine Trl Suite 102 Smyrna TN, 37167About
Dr. Krystal Hay is a physiatrist practicing in Smyrna, TN. Dr. Hay is a medical doctor specializing in physical medicine and rehabilitation. As a physiatrist, Dr. Hay focuses on a patients ability to function, and can treat multiple conditions that affect the brain, nerves, spine, bones, muscles, joints, ligaments and tendons. Dr. Hay can diagnose and treat pain that is a result of injury, disease or a disabling condition. Physiatrists often lead a team of physical therapists, occupational therapists and physicians in a patients treatment or prevention plan.
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Expert Publications
Data provided by the National Library of Medicine- Inhibition of herpes simplex virus reactivation by dipyridamole.
- Spleen cell-mediated cytotoxicity of hamster cells transformed by Herpes simplex virus: evidence for virus-specific membrane antigen.
- Spleen cell-mediated cytotoxicity of hamster cells transformed by Herpes simplex virus: evidence for virus-specific membrane antigen.
- Expression of herpes simplex virus type 2 latency-associated transcript in neurons and nonneurons.
- Immunological studies with hamster cells transformed by cytomegalovirus and herpes simplex virus type 1.
- Immunological studies with hamster cells transformed by cytomegalovirus and herpes simplex virus type 1.
- Latency-associated transcript but not reactivatable virus is present in sensory ganglion neurons after inoculation of thymidine kinase-negative mutants of herpes simplex virus type 1.
- Herpes simplex latent infection: quantitation of latency-associated transcript-positive neurons and reactivable neurons.
- Herpes simplex virus latent infection: reactivation and elimination of latency after neurectomy.
- Expression of type-common envelope antigens by herpes simplex virus type 2-transformed hamster cells.
- Secondary herpes simplex virus latent infection in transplanted ganglia.
- Neuronal control of herpes simplex virus latency.
- Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside.
- Uninhibited growth and metastases of herpes simplex virus-transformed cells in virus-sensitized hosts.
- Uninhibited growth and metastases of herpes simplex virus-transformed cells in virus-sensitized hosts.
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