Dr. David Eric Symer MD, PHD
Hematologist (Blood Specialist) | Hematology
300 W 10th Ave Columbus OH, 43210About
Dr. David Symer is a hematology internist practicing in Columbus, OH. Dr. Symer specializes in cases of suspected blood disorders when a diagnosis is unclear and further medical care is needed. Hematology internists work with other specialists such as surgeons, radiation therapists and gynecologists in order to get a better understanding on the cases. Often, patients are asked to participate in clinical research that could result in more efficient treatment methods.
Education and Training
Johns Hopkins University School of Medicine,Baltimore, Md, United States MD 1993
Johns Hopkins University School of Medicine 1993
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Evidence for a causal association between human papillomavirus and a subset of head and neck cancers.
- Genomic stability. Hip-hopping out of control.
- Human l1 retrotransposition is associated with genetic instability in vivo.
- Inhibition or activation of human T cell receptor transfectants is controlled by defined, soluble antigen arrays.
- Extensive variation between inbred mouse strains due to endogenous L1 retrotransposition.
- Lysine methylation of HIV-1 Tat regulates transcriptional activity of the viral LTR.
- MouseIndelDB: a database integrating genomic indel polymorphisms that distinguish mouse strains.
- Mutations in U4atac snRNA, a component of the minor spliceosome, in the developmental disorder MOPD I.
- The molecular and cellular basis of variable craniofacial phenotypes and their genetic rescue in Twisted gastrulation mutant mice.
- Mouse endogenous retroviruses can trigger premature transcriptional termination at a distance.
- The bonobo genome compared with the chimpanzee and human genomes.
- How do mammalian transposons induce genetic variation? A conceptual framework: the age, structure, allele frequency, and genome context of transposable elements may define their wide-ranging biological impacts.
- SRGAP1 is a candidate gene for papillary thyroid carcinoma susceptibility.
- Ultra-rare mutation in long-range enhancer predisposes to thyroid carcinoma with high penetrance.
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