Dr. Michael Werner nikolaus Deininger MD
Hematologist-Oncologist | Hematology & Oncology
1950 Circle Of Hope Dr Salt Lake City UT, 84112About
Dr. Michael Deininger is a hematologist oncologist practicing in Salt Lake City, UT. Dr. Deininger specializes in the diagnosis, treatment and prevention of blood diseases such as anemia, hemophilia, sickle-cell disease, leukemia and lymphoma. Hematologist Oncologists are also trained in the study of cancer and its attack on other organs.
Education and Training
Julius-Maximilians-Universit‰t W¸rzburg Medizinische Fakult‰t 1994
Provider Details
Expert Publications
Data provided by the National Library of Medicine- BCR-ABL tyrosine kinase activity regulates the expression of multiple genes implicated in the pathogenesis of chronic myeloid leukemia.
- Selection and characterization of BCR-ABL positive cell lines with differential sensitivity to the tyrosine kinase inhibitor STI571: diverse mechanisms of resistance.
- The molecular biology of chronic myeloid leukemia.
- Transcription factor BACH2 is transcriptionally regulated by the BCR/ABL oncogene.
- High Bad and Bax mRNA expression correlate with negative outcome in acute myeloid leukemia (AML).
- Deletions of the derivative chromosome 9 do not account for the poor prognosis associated with Philadelphia-positive acute lymphoblastic leukemia.
- CML with an e1a3 BCR-ABL fusion: rare, benign, and a potential diagnostic pitfall.
- The BCR/ABL-extra signal fluorescence in situ hybridization system reliably detects deletions upstream of the ABL locus: implications for reporting of results and followup of chronic myelogenous leukemia patients.
- High levels of BAX, low levels of MRP-1, and high platelets are independent predictors of response to imatinib in myeloid blast crisis of CML.
- Emergence of clonal cytogenetic abnormalities in Ph- cells in some CML patients in cytogenetic remission to imatinib but restoration of polyclonal hematopoiesis in the majority.
- Several Bcr-Abl kinase domain mutants associated with imatinib mesylate resistance remain sensitive to imatinib.
- Imatinib improves but may not fully reverse the poor prognosis of patients with CML with derivative chromosome 9 deletions.
- Imatinib normalizes bone marrow vascularity in patients with chronic myeloid leukemia in first chronic phase.
- Imatinib in patients with newly diagnosed chronic-phase chronic myeloid leukemia.
- Cytogenetic studies in patients on imatinib.
Clinical Trials
Treatments
- Acute Myeloid Leukemia (aml)
- Lymphoma
- Multiple Myeloma
- Leukemia
- Dermal Aesthetics
- Oral Surgery Procedures
- Extra Corporeal Shockwave Therapy
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Nearby Providers
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- Dr. Justin A Call M.D.3590 W 9000 S West Jordan UT 84088
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- Dr. Scott James Samuelson M.D.333 S 900 E Salt Lake City UT 84102