Mr. David R Howlett MD
Family Practitioner
13 Church Rd East Granby CT, 06026About
Dr. David Howlett is a family practitioner practicing in East Granby, CT. Dr. Howlett specializes in comprehensive health care for people of all ages. In addition to diagnosing and treating illnesses, family practitioners also put focus on preventative care with routine checkups, tests and personalized coaching on how to maintain a healthy lifestyle. Dr. Howlett possesses immense general knowledge on maintaining health and today, family practitioners provide more care for the underserved and rural populations than any other medical specialty.
Education and Training
Univ Of Ct Sch Of Med- Farmington Ct 1980
University of Connecticut School of Medicine 1983
Board Certification
Family MedicineAmerican Board of Family MedicineABFM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Alzheimer's disease: correlation of the suppression of beta-amyloid peptide secretion from cultured cells with inhibition of the chymotrypsin-like activity of the proteasome.
- Computationally derived structural models of the beta-amyloid found in Alzheimer's disease plaques and the interaction with possible aggregation inhibitors.
- Fractionation and characterization of oligomeric, protofibrillar and fibrillar forms of beta-amyloid peptide.
- In search of an enzyme: the beta-secretase of Alzheimer's disease is an aspartic proteinase.
- Neurodegenerative changes associated with beta-amyloid deposition in the brains of mice carrying mutant amyloid precursor protein and mutant presenilin-1 transgenes.
- Urinary excretion of the sulphate and glucuronide of 3-methoxy-4-hydroxyphenylethyleneglycol in a manic-depressive patient.
- Production of beta-amyloid by primary human foetal mixed brain cell cultures and its modulation by exogenous soluble beta-amyloid.
- Hyperphosphorylated tau and paired helical filament-like structures in the brains of mice carrying mutant amyloid precursor protein and mutant presenilin-1 transgenes.
- Cognitive correlates of Abeta deposition in male and female mice bearing amyloid precursor protein and presenilin-1 mutant transgenes.
- Imaging and spatial distribution of beta-amyloid peptide and metal ions in Alzheimer's plaques by laser ablation-inductively coupled plasma-mass spectrometry.
- MAPK-activated protein kinase 2 deficiency in microglia inhibits pro-inflammatory mediator release and resultant neurotoxicity. Relevance to neuroinflammation in a transgenic mouse model of Alzheimer disease.
- Abeta(1-42) modulation of Akt phosphorylation via alpha7 nAChR and NMDA receptors.
- The pathology of APP transgenic mice: a model of Alzheimer's disease or simply overexpression of APP?
- Neurochemical changes in a double transgenic mouse model of Alzheimer's disease fed a pro-oxidant diet.
- Association of plasma clusterin concentration with severity, pathology, and progression in Alzheimer disease.
Treatments
- Diabetes
- High Cholesterol
- Actinic Keratosis
- Type 2 Diabetes
- Pain
- Vascular Disease
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