Dr. George Richard Uhl MD PHD
Neurologist | Neurology
4940 Eastern Ave Dept Of Neurology Baltimore MD, 21224About
Dr. George Uhl is a distinguished Neurologist in Baltimore, MD. Dr. Uhl specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Uhl employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Uhl is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
Johns Hopkins Univ Sch of Med, Baltimore Md 1979
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Absence of G-protein activation by mu-opioid receptor agonists in the spinal cord of mu-opioid receptor knockout mice.
- Visceral chemical nociception in mice lacking mu-opioid receptors: effects of morphine, SNC80 and U-50,488.
- The mu opiate receptor as a candidate gene for pain: polymorphisms, variations in expression, nociception, and opiate responses.
- Dopamine transporter: transmembrane phenylalanine mutations can selectively influence dopamine uptake and cocaine analog recognition.
- Heterozygous VMAT2 knockout mice display prolonged QT intervals: possible contributions to sudden death.
- Cocaine reward and MPTP toxicity: alteration by regional variant dopamine transporter overexpression.
- The mu-opioid receptor gene-dose dependent reductions in G-protein activation in the pons/medulla and antinociception induced by endomorphins in mu-opioid receptor knockout mice.
- Dopamine transporter proline mutations influence dopamine uptake, cocaine analog recognition, and expression.
- Dopamine transporter transmembrane domain polar mutants: DeltaG and DeltaDeltaG values implicate regions important for transporter functions.
- Stress-induced analgesia in mu-opioid receptor knockout mice reveals normal function of the delta-opioid receptor system.
- Human dopamine transporter gene: coding region conservation among normal, Tourette's disorder, alcohol dependence and attention-deficit hyperactivity disorder populations.
- Long forms of the dopamine receptor (DRD4) gene VNTR are more prevalent in substance abusers: no interaction with functional alleles of the catechol-o-methyltransferase (COMT) gene.
- Dopamine transporter tryptophan mutants highlight candidate dopamine- and cocaine-selective domains.
- The VMAT2 gene in mice and humans: amphetamine responses, locomotion, cardiac arrhythmias, aging, and vulnerability to dopaminergic toxins.
- Ethanol consumption and reward are decreased in mu-opiate receptor knockout mice.
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George Uhl
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