Dr. Leonard G. Meggs M.D.
Nephrologist (Kidney Specialist) | Nephrology
1514 Jefferson Highway New Orleans LA, 70121About
Dr. Leonard Meggs is a nephrologist practicing in New Orleans, LA. Dr. Meggs specializes in the care and treatment of the kidneys. As a nephrologist, Dr. Meggs most typically treats conditions like kidney stones, chronic kidney disease, acute renal failure, polycystuc kidney disease, high blood pressure and more. Nephrologists are also experts on kidney transplantation and dialysis. They are usually referred to by primary care physicians for problems related to the kidneys, and while they can perform tests to diagnose kidney disorders, they do not perform surgeries.
Education and Training
Yale School of Medicine 1975
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Overexpression of insulin-like growth factor-1 attenuates the myocyte renin-angiotensin system in transgenic mice.
- Emerging concepts in antihypertensive therapy: the benefits of angiotensin II blockade.
- Angiotensin II promotes glucose-induced activation of cardiac protein kinase C isozymes and phosphorylation of troponin I.
- Molecular biology of protein kinase C signaling in cardiac myocytes.
- High glucose promotes mesangial cell apoptosis by oxidant-dependent mechanism.
- IGF-1 inhibits the mitochondrial apoptosis program in mesangial cells exposed to high glucose.
- Glycation end-product cross-link breaker reduces collagen and improves cardiac function in aging diabetic heart.
- Chronic coronary arterial stenosis impairs alpha 1-adrenoreceptor signaling and cardiac performance in rats.
- Re-expression of alpha skeletal actin and regulation of alpha 1 adrenoceptor signalling in DOCA-salt hypertension in rats.
- PKCepsilon inhibits the hyperglycemia-induced apoptosis signal in adult rat ventricular myocytes.
- PKC-{epsilon}-dependent survival signals in diabetic hearts.
- Activated IGF-1R inhibits hyperglycemia-induced DNA damage and promotes DNA repair by homologous recombination.
- Chronic nonocclusive coronary artery constriction in rats. Beta-adrenoceptor signal transduction and ventricular failure.
- Inhibition of wild-type p66ShcA in mesangial cells prevents glycooxidant-dependent FOXO3a regulation and promotes the survival phenotype.
- Cellular basis of chronic ventricular remodeling after myocardial infarction in rats.
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