Dr. Alice Moran practices Periodontics in San Clemente, CA. A periodontist is a dentist who specializes in the prevention, diagnosis, and treatment of periodontal disease, and in the placement of dental implants. Dr. Moran has expertise in the treatment of oral inflammation and often treats problematic periodontal cases, such as those with severe gum disease or a complex medical history. Some treatments that Dr. Moran provides are scaling and root planing and root surface debridement.
- Serotyping of Campylobacter jejuni based on heat-stable antigens: relevance, molecular basis and implications in pathogenesis.
- Differentiation of Helicobacter pylori isolates based on lectin binding of cell extracts in an agglutination assay.
- Lack of a relationship between Lewis antigen expression and cagA, CagA, vacA and VacA status of Irish Helicobacter pylori isolates.
- Human monoclonal immunoglobulin M antibodies to ganglioside GM1 show diverse cross-reactivities with lipopolysaccharides of Campylobacter jejuni strains associated with Guillain-Barré syndrome.
- A structural comparison of lipopolysaccharides from two strains of Helicobacter pylori, of which one strain (442) does and the other strain (471) does not stimulate pepsinogen secretion.
- Structural studies on lipopolysaccharides of serologically non-typable strains of Helicobacter pylori, AF1 and 007, expressing Lewis antigenic determinants.
- Ganglioside-induced antiganglioside antibodies from a neuropathy patient cross-react with lipopolysaccharides of Campylobacter jejuni associated with Guillain-Barré syndrome.
- Lipopolysaccharide structures of Helicobacter pylori genomic strains 26695 and J99, mouse model H. pylori Sydney strain, H. pylori P466 carrying sialyl Lewis X, and H. pylori UA915 expressing Lewis B classification of H. pylori lipopolysaccharides in
- Relationship of blood group determinants on Helicobacter pylori lipopolysaccharide with host lewis phenotype and inflammatory response.
- Helicobacter pylori lipopolysaccharide-mediated gastric and extragastric pathology.
- Biological activities of lipopolysaccharides are determined by the shape of their
- Lewis X structures in the O antigen side-chain promote adhesion of Helicobacter pylori to the gastric epithelium.
- Structure of an atypical O-antigen polysaccharide of Helicobacter pylori containing a novel monosaccharide 3-C-methyl-D-mannose.
- Genotyping of cagA and vacA, Lewis antigen status, and analysis of the poly-(C) tract in the alpha(1,3)-fucosyltransferase gene of Irish Helicobacter pylori isolates.
- Cytotoxicity associated with induction of nitric oxide synthase in rat duodenal epithelial cells in vivo by lipopolysaccharide of Helicobacter pylori: inhibition by superoxide dismutase.
- Comparison of three serological methods for detection of Lewis antigens on the surface of Helicobacter pylori.
- Sialylation of lipooligosaccharide cores affects immunogenicity and serum resistance of Campylobacter jejuni.
- Why Helicobacter pylori has Lewis antigens.
- The relationship between O-chain expression and colonisation ability of Helicobacter pylori in a mouse model.
- Chemical structure of the core oligosaccharide of aerotolerant Campylobacter jejuni O:2 lipopolysaccharide.
- Complement activation directly induced by Helicobacter pylori.
- Development of an immunoassay for rapid detection of ganglioside GM(1) mimicry in Campylobacter jejuni strains.
- Complement-mediated lipopolysaccharide release and outer membrane damage in Escherichia coli J5: requirement for C9.
- Structure of a D-glycero -D-manno-heptan from the lipopolysaccharide of
- Evaluation of clarithromycin resistance and cagA and vacA genotyping of Helicobacter pylori strains from the west of Ireland using line probe assays.
- Effect of purified lipopolysaccharides from strains of Helicobacter pylori and Helicobacter felis on acid secretion in mouse gastric glands in vitro.
- Helicobacter pyroli, Lewis antigens, and inflammation.
- Helicobacter pylori lipopolysaccharide-provoked injury to rat gastroduodenal microvasculature involves inducible nitric oxide synthase.
- Relationship of anti-Lewis x and anti-Lewis y antibodies in serum samples from gastric cancer and chronic gastritis patients to Helicobacter pylori-mediated autoimmunity.
- Lipopolysaccharides in the development of the Guillain-Barré syndrome and Miller Fisher syndrome forms of acute inflammatory peripheral neuropathies.
- Helicobacter pylori lipopolysaccharide provokes iNOS-mediated acute systemic microvascular inflammatory responses in rat cardiac, hepatic, renal and pulmonary tissues.
- Attenuation of Helicobacter pylori endotoxin-provoked rat intestinal inflammation by selective inhibition of the inducible nitric oxide synthase.
- Detection and identification of Helicobacter pylori directly from gastric biopsies using polymerase chain reaction.
- Separation of two lipopolysaccharide populations with different contents of O-antigen factor 122 in Salmonella enterica serovar typhimurium.
- Application of methylation analysis in the determination of the structure of disaccharides containing 2,3-diamino-2,3-dideoxy-D-glucose (GlcN3N) associated with the backbone of lipid A.
- Low biological activity of Helicobacter pylori lipopolysaccharide.
- Separation and characterization of two chemically distinct lipopolysaccharides in two Pectinatus species.
- Compositional analysis of Helicobacter pylori rough-form lipopolysaccharides.
- Triggering receptor expressed on myeloid cells-1 (TREM-1) expression on gastric epithelium: implication for a role of TREM-1 in Helicobacter pylori infection.
- Complete upper airway obstruction, a misquote too far?
- Chemical characterization of Campylobacter jejuni lipopolysaccharides containing N-acetylneuraminic acid and 2,3-diamino-2,3-dideoxy-D-glucose.
- Structural analysis of the lipid A component of Campylobacter jejuni CCUG 10936
- Helicobacter pylori lipopolysaccharide activity in human peripheral blood mononuclear leukocyte cultures.
- Sweet-talk: role of host glycosylation in bacterial pathogenesis of the gastrointestinal tract.
- A comparative study of the rod and coccoid forms of Campylobacter jejuni ATCC 29428.
- Interaction of Campylobacter jejuni with extracellular matrix components.
- Production and characterization of a compound inhibitory to Vibrio parahaemolyticus from Proteus vulgaris.
- Serological analysis of the heat-stable antigens involved in serotyping Campylobacter jejuni and Campylobacter coli.
- Effect of medium supplements, illumination and superoxide dismutase on the production of coccoid forms of Campylobacter jejuni ATCC 29428.
- Factors affecting production of coccoid forms by Campylobacter jejuni on solid media during incubation.
- Chemical structures of the core region of Campylobacter jejuni O:3 lipopolysaccharide and an associated polysaccharide.
- Structures of the O-specific polysaccharide chains of Pectinatus cerevisiiphilus and Pectinatus frisingensis lipopolysaccharides.
- Sequence of the gene encoding the 16S rRNA of the beer spoilage organism Megasphaera cerevisiae.
- Biological and serological characterization of Campylobacter jejuni lipopolysaccharides with deviating core and lipid A structures.
- Cell surface characteristics of Helicobacter pylori.
- Neutrophil activation by Helicobacter pylori lipopolysaccharides.
- Interaction of lipopolysaccharides of Helicobacter pylori with basement membrane protein laminin.
- Biochemical characterization of Campylobacter fetus lipopolysaccharides.
- Chemical structure of the lipid A component of lipopolysaccharides of the genus Pectinatus.
- Chemical structures of the core regions of Campylobacter jejuni serotypes O:1, O:4, O:23, and O:36 lipopolysaccharides.
- Chemical structures of the core regions of Campylobacter jejuni serotypes O:1, O:4, O:23, and O:36 lipopolysaccharides.
- Chemical structure of the core region of Campylobacter jejuni serotype O:2 lipopolysaccharide.
- Antibodies to Campylobacter jejuni react with myelin proteins of the central nervous system.
- Lipopolysaccharide of the Helicobacter pylori type strain NCTC 11637 (ATCC 43504): structure of the O antigen chain and core oligosaccharide regions.
- The O-specific polysaccharide chain of Campylobacter fetus serotype B lipopolysaccharide is a D-rhamnan terminated with 3-O-methyl-D-rhamnose (D-acofriose).
- Pathogenic properties of Helicobacter pylori.
- The role of lipopolysaccharide in Helicobacter pylori pathogenesis.
- Typing of Helicobacter pylori with monoclonal antibodies against Lewis antigens in lipopolysaccharide.
- Biological characterization of Campylobacter fetus lipopolysaccharides.
- Effect of Helicobacter pylori lipopolysaccharide (LPS) and LPS derivatives on the production of tissue factor and plasminogen activator inhibitor type 2 by human blood mononuclear cells.
- Bacterial surface structures--an update.
- Interactions of bacterial adhesins with extracellular matrix and plasma proteins: pathogenic implications and therapeutic possibilities.
- Guidelines for the management of Helicobacter pylori-related upper gastrointestinal diseases. Irish Helicobacter Pylori Study Group.
- Identification of the N-acetylneuraminyllactose-specific laminin-binding protein of Helicobacter pylori.
- Molecular mimicry between Helicobacter pylori and the host.
- The O-specific polysaccharide chain of Campylobacter fetus serotype A lipopolysaccharide is a partially O-acetylated 1,3-linked alpha-D-mannan.
- Uptake on Helicobacter pylori research. Pathogenesis and host response.
- Pathogenesis of gastric Helicobacter pylori.
- Influence of medium composition on the growth and antigen expression of Helicobacter pylori.
- Role of adherence in interleukin-8 induction in Helicobacter pylori-associated gastritis.
- Serum antibodies against gangliosides and Campylobacter jejuni lipopolysaccharides in Miller Fisher syndrome.
- Structural characterization of the lipid A component of Helicobacter pylori rough- and smooth-form lipopolysaccharides.
- Structure and conserved characteristics of Campylobacter jejuni
- Helicobacter pylori expresses Lewis X.
- Coccoid forms of Helicobacter pylori.
- Simultaneous expression of type 1 and type 2 Lewis blood group antigens by Helicobacter pylori lipopolysaccharides. Molecular mimicry between h. pylori lipopolysaccharides and human gastric epithelial cell surface glycoforms.
- Unique structural and biological features of Helicobacter pylori lipopolysaccharides.
- Helper T-cell subsets, interleukins, gastritis and Helicobacter pylori.
- Lipopolysaccharides from Campylobacter jejuni O:41 strains associated with Guillain-Barré syndrome exhibit mimicry of GM1 ganglioside.
- C7 deficiency in an Irish family: a deletion defect which is predominant in the Irish.
- Mimicry of blood group antigen A by Helicobacter mustelae and H. pylori.
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