Mr. Terry Wayne Wright DMD PSC
Periodontist | Periodontics
118 Caroline Avenue Suite 2 Pikeville KY, 41502About
Dr. Terry Wright practices Periodontics in Pikeville, KY. A periodontist is a dentist who specializes in the prevention, diagnosis, and treatment of periodontal disease, and in the placement of dental implants. Dr. Wright has expertise in the treatment of oral inflammation and often treats problematic periodontal cases, such as those with severe gum disease or a complex medical history. Some treatments that Dr. Wright provides are scaling and root planing and root surface debridement.
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Expert Publications
Data provided by the National Library of Medicine- Passive intranasal monoclonal antibody prophylaxis against murine Pneumocystis carinii pneumonia.
- Neither neutrophils nor reactive oxygen species contribute to tissue damage during Pneumocystis pneumonia in mice.
- Activity of Hoechst 33258 against Pneumocystis carinii f. sp. muris, Candida albicans, and Candida dubliniensis.
- Pneumocystis carinii activates the NF-kappaB signaling pathway in alveolar epithelial cells.
- Inhibition of surfactant activity by Pneumocystis carinii organisms and components in vitro.
- Complement and Fc function are required for optimal antibody prophylaxis against Pneumocystis carinii pneumonia.
- Active immunization against Pneumocystis carinii with a recombinant P. carinii antigen.
- p21Cip1 protection against hyperoxia requires Bcl-XL and is uncoupled from its ability to suppress growth.
- Immunopathogenesis of Pneumocystis carinii pneumonia.
- Contribution of T cell subsets to the pathophysiology of Pneumocystis-related immunorestitution disease.
- Sensitized CD8+ T cells fail to control organism burden but accelerate the onset of lung injury during Pneumocystis carinii pneumonia.
- Pneumocystis stimulates MCP-1 production by alveolar epithelial cells through a JNK-dependent mechanism.
- Parenchymal cell TNF receptors contribute to inflammatory cell recruitment and respiratory failure in Pneumocystis carinii-induced pneumonia.
- Anti-CD3 antibody decreases inflammation and improves outcome in a murine model of Pneumocystis pneumonia.
- Mixed Lineage Kinase 3 deficiency delays viral clearance in the lung and is associated with diminished influenza-induced cytopathic effect in infected cells.
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