Dr. Rachel Aubrey Gravel M.D.
Pediatrician
2200 Bergquist Dr Lackland TX, 78236About
Dr. Rachel Gravel is a pediatrician practicing in Lackland, TX. Dr. Gravel is a doctor who specializes in the health care of children. As a pedicatrician, Dr. Gravel diagnoses and treats infections, injuries, diseases and other disorders in children. Pediatricians typically work with infants, children, teenagers and young adults up to age 21. They practice medical care as well as preventative health care. Dr. Gravel can oversee and manage the physical, mental and emotional health of their patients.
Board Certification
PediatricsAmerican Board of PediatricsABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Neuronal apoptosis in sudden infant death syndrome.
- Mechanism of biotin responsiveness in biotin-responsive multiple carboxylase deficiency.
- Genetic polymorphisms in methylenetetrahydrofolate reductase and methionine synthase, folate levels in red blood cells, and risk of neural tube defects.
- Sialidase-mediated depletion of GM2 ganglioside in Tay-Sachs neuroglia cells.
- A common variant in methionine synthase reductase combined with low cobalamin (vitamin B12) increases risk for spina bifida.
- Molecular basis for methionine synthase reductase deficiency in patients belonging to the cblE complementation group of disorders in folate/cobalamin metabolism.
- Overview of mutations in the PCCA and PCCB genes causing propionic acidemia.
- Molecular cloning, expression and physical mapping of the human methionine synthase reductase gene.
- Genetic complementation in heterokaryons of human fibroblasts defective in cobalamin metabolism.
- I. Abnormalities in cells of the testis, efferent ducts, and epididymis in juvenile and adult mice with beta-hexosaminidase A and B deficiency.
- II. Characterization and development of the regional- and cellular-specific abnormalities in the epididymis of mice with beta-hexosaminidase A deficiency.
- A common polymorphism in methionine synthase reductase increases risk of premature coronary artery disease.
- Expression in Escherichia coli of N- and C-terminally deleted human holocarboxylase synthetase. Influence of the N-terminus on biotinylation and identification of a minimum functional protein.
- Biotin-response organicaciduria. Multiple carboxylase defects and complementation studies with propionicacidemia in cultured fibroblasts.
- Biotin-response organicaciduria. Multiple carboxylase defects and complementation studies with propionicacidemia in cultured fibroblasts.
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