Dr. Michael F Beers MD
Pulmonologist | Pulmonary Disease
3400 Spruce Street Philadelphia PA, 19104About
Dr. Michael Beers practices Pulmonology in Philadelphia, PA. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Beers manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Education and Training
Thomas Jefferson University Jefferson Medical College MD
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Critical Care Medicine
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Inhibition of lung surfactant protein B expression during Pneumocystis carinii pneumonia in mice.
- Structural requirements for intracellular targeting of SP-C proprotein.
- P. carinii induces selective alterations in component expression and biophysical
- Very low surfactant protein C contents in newborn Belgian White and Blue calves with respiratory distress syndrome.
- Biosynthesis of surfactant protein C: characterization of aggresome formation by EGFP chimeras containing propeptide mutants lacking conserved cysteine residues.
- Post-translational processing of surfactant protein-C proprotein: targeting motifs in the NH(2)-terminal flanking domain are cleaved in late compartments.
- Pneumocystis carinii pneumonia alters expression and distribution of lung collectins SP-A and SP-D.
- Aspergillus fumigatus-induced allergic airway inflammation alters surfactant homeostasis and lung function in BALB/c mice.
- Maintenance of differentiated function of the surfactant system in human fetal lung type II epithelial cells cultured on plastic.
- Synthesis and post-translational processing of surfactant protein C.
- Biosynthesis of surfactant protein C (SP-C). Sorting of SP-C proprotein involves homomeric association via a signal anchor domain.
- Involvement of cathepsin H in the processing of the hydrophobic
- Total extracellular surfactant is increased but abnormal in a rat model of gram-negative bacterial pneumonia.
- The late asthmatic response is linked with increased surface tension and reduced surfactant protein B in mice.
- Deletion of exon 4 from human surfactant protein C results in aggresome formation and generation of a dominant negative.
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