Dr. William Joseph Muller MD, PHD
Infectious Disease Specialist (Pediatric) | Pediatric Infectious Diseases
2300 N Childrens Plz Box 20 Chicago IL, 60614About
Dr. William Muller is a pediatric infectious disease specialist practicing in Chicago, IL. Dr. Muller specializes in recurring or persistent diseases caused by bacteria, parasites or fungus in infants, children and adolescents. Pediatric infectious disease specialists also provide consultation to other health care professionals dealing with complex cases.
Board Certification
PediatricsAmerican Board of PediatricsABP- Pediatric Infectious Diseases
Provider Details
Expert Publications
Data provided by the National Library of Medicine- The catalytic activity of the ErbB-2 receptor tyrosine kinase is essential for embryonic development.
- Centrosome abnormalities, recurring deletions of chromosome 4, and genomic amplification of HER2/neu define mouse mammary gland adenocarcinomas induced by mutant HER2/neu.
- Genetic instability favoring transversions associated with ErbB2-induced mammary tumorigenesis.
- Kinase suppressor of Ras (KSR) is a scaffold which facilitates mitogen-activated protein kinase activation in vivo.
- ErbB2 is required for muscle spindle and myoblast cell survival.
- General keynote: expression of epidermal growth factor receptor family in transgenic mouse models of human breast cancer.
- Estradiol promotes growth and angiogenesis in polyoma middle T transgenic mouse mammary tumor explants.
- Transforming growth factor beta signaling impairs Neu-induced mammary
- Gene expression profiling of neu-induced mammary tumors from transgenic mice reveals genetic and morphological similarities to ErbB2-expressing human breast cancers.
- Angiogenesis impairment in Id-deficient mice cooperates with an Hsp90 inhibitor to completely suppress HER2/neu-dependent breast tumors.
- Progression to malignancy in the polyoma middle T oncoprotein mouse breast cancer model provides a reliable model for human diseases.
- Ets2-dependent stromal regulation of mouse mammary tumors.
- Changes in the expression of many Ets family transcription factors and of potential target genes in normal mammary tissue and tumors.
- Epidermal growth factor receptor-dependent activation of Gab1 is involved in ErbB-2-mediated mammary tumor progression.
- Angiogenic acceleration of Neu induced mammary tumor progression and metastasis.
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