Rajesh Kumar Dubey M.D.
Cardiologist | Cardiovascular Disease
1401 Spanos Ct Suite 230 Modesto CA, 95355About
Dr. Rajesh Dubey is a cardiologist practicing in Modesto, CA. Dr. Dubey specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Dubey also practices preventative medicine, helping patients maintain a heart-healthy life.
Education and Training
University of Calcutta / Medical College MD
Calcutta National Medical College 1991
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Angiotensin receptor subtype 1 mediates angiotensin II enhancement of isoproterenol-induced cyclic AMP production in preglomerular microvascular smooth muscle cells.
- Role of adenosine A(1) receptors in modulating extracellular adenosine levels.
- Xeno-oestrogens and phyto-oestrogens induce the synthesis of leukaemia inhibitory factor by human and bovine oviduct cells.
- Estradiol inhibits smooth muscle cell growth in part by activating the cAMP-adenosine pathway.
- A(2B) receptors mediate antimitogenesis in vascular smooth muscle cells.
- Purification and characterisation of gelonin from seeds of Gelonium multiflorum.
- Sustained reduction in myocardial reperfusion injury with an adenosine receptor antagonist: possible role of the neutrophil chemoattractant response.
- Clinically used estrogens differentially inhibit human aortic smooth muscle cell growth and mitogen-activated protein kinase activity.
- Significance of charge on lysine residue of ovine luteinizing hormone on immunological and biological properties of the hormone.
- Cellular and biochemical mechanisms by which environmental oestrogens influence reproductive function.
- Cardiac fibroblasts express the cAMP-adenosine pathway.
- Methoxyestradiols mediate the antimitogenic effects of estradiol on vascular smooth muscle cells via estrogen receptor-independent mechanisms.
- Estrogen-induced cardiorenal protection: potential cellular, biochemical, and molecular mechanisms.
- Estradiol metabolites inhibit endothelin synthesis by an estrogen receptor-independent mechanism.
- Effects of estradiol and its metabolites on glomerular endothelial nitric oxide synthesis and mesangial cell growth.
Treatments
- Heart Disease
- Type 2 Diabetes
- Coronary Artery Disease (cad)
- Pain
- Vascular Disease
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