Dr. Donald A Kennerly MD
Internist
4004 Worth St Suite 300 Dallas TX, 75246About
Dr. Donald Kennerly is an internist practicing in Dallas, TX. Dr. Kennerly specializes in the medical treatment of adults. Internists can act as a primary physician or a consultant to a primary physician. They manage both common and rare diseases. Dr. Kennerly provides comprehensive care and manages treatment with surgeons as well. Internists establish long-term relationships with their patients and incorporate disease prevention and mental health care into their practice.
Education and Training
Washington Univ Sch Of Med- St Louis Mo 1980
Washington Center / School of Medicine 1980
Washington University in St. Louis School of Medicine 1980
Board Certification
Allergy and ImmunologyAmerican Board of Allergy and ImmunologyABAI
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Comparison of diglyceride production from choline-containing phosphoglycerides in human neutrophils stimulated with N-formylmethionyl-leucylphenylalanine, ionophore A23187 or phorbol 12-myristate 13-acetate.
- Nutrition support in the intensive care unit. Adequacy, timeliness, and outcomes.
- Impact of the national asthma guidelines on internal medicine primary care and specialty practice.
- Assessment of receptor-dependent activation of phosphatidylcholine hydrolysis by both phospholipase D and phospholipase C.
- Quantitative analysis of water-soluble products of cell-associated phospholipase
- Isolation and enzymic assay of choline and phosphocholine present in cell extracts with picomole sensitivity.
- Large-scale deployment of the Global Trigger Tool across a large hospital system: refinements for the characterisation of adverse events to support patient safety learning opportunities.
- An indirect pathway of receptor-mediated 1,2-diacylglycerol formation in mast cells. I. IgE receptor-mediated activation of phospholipase D.
- Phosphatidylcholine is a quantitatively more important source of increased 1,2-diacylglycerol than is phosphatidylinositol in mast cells.
- Modulation of stimulated phospholipid metabolism in mast cells by pharmacologic agents that increase cyclic 3',5' adenosine monophosphate levels.
- Modulation of stimulated phospholipid metabolism in mast cells by pharmacologic agents that increase cyclic 3',5' adenosine monophosphate levels.
- Use of diacylclycerol kinase to quantitate picomole levels of 1,2-diacylglycerol.
- Use of diacylclycerol kinase to quantitate picomole levels of 1,2-diacylglycerol.
- Description and evaluation of adaptations to the global trigger tool to enhance value to adverse event reduction efforts.
- Characterization of adverse events detected in a large health care delivery system using an enhanced global trigger tool over a five-year interval.
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