Dr. Bryce Anthony Binstadt M.D., PH.D.
Rheumatologist (Pediatric) | Pediatric Rheumatology420 Delaware St Se Mmc 817 Minneapolis MN, 55455
Dr. Bryce Binstadt practices Pediatric Rheumatology in Minneapolis, MN. Pediatric rheumatologists treat child patients who suffer from pain in the musculoskeletal system, have symptoms of arthritis, or an autoimmune disorder. Dr. Binstadt evaluates and treats a variety of joint, muscle, and bone disorders; including autoimmune disorders, such as lupus, juvenile rheumatoid arthritis, scleroderma, Kawasaki disease, post-infectious arthritis, chronic vasculitis, and inflammatory disorders of the muscle, eye, or other organs. Dr. Binstadt also is trained in the evaluation of prolonged fever, unexplained complaints of chronic musculoskeletal pain, weakness, poor appetite, fatigue, and/or loss of function or skills.
Education and Training
Mayo Med Sch, Rochester Mn 1999
PediatricsAmerican Board of PediatricsABP- Pediatric Rheumatology
DermatologyAmerican Board of DermatologyABD
- Use of mutant Jurkat T cell lines to define human natural killer cell surface receptor signaling pathways.
- Celiac disease presenting with chilblains in an adolescent girl.
- Mutations of the hemophagocytic lymphohistiocytosis-associated gene UNC13D in a
- The same systemic autoimmune disease provokes arthritis and endocarditis via distinct mechanisms.
- Cardiac operations for North American children with rheumatic diseases: 1985-2005.
- Tyrosine phosphorylation provides an early and requisite signal for the activation of natural killer cell cytotoxic function.
- Malar rash in systemic juvenile idiopathic arthritis.
- Pericarditis as the presenting feature of adrenoleukodystrophy.
- Absence of β2 integrins impairs regulatory T cells and exacerbates CD4+ T cell-dependent autoimmune carditis.
- The neuropeptide neuromedin U promotes autoantibody-mediated arthritis.
- Incomplete TCR-β allelic exclusion accelerates spontaneous autoimmune arthritis in K/BxN TCR transgenic mice.
- Autoantibody-mediated arthritis in the absence of C3 and activating Fcγ receptors: C5 is activated by the coagulation cascade.
- Autoimmune retinopathy in a patient with autoimmune polyendocrine syndrome type I.
- Macrophage scavenger receptor 1 (Msr1, SR-A) influences B cell autoimmunity by regulating soluble autoantigen concentration.
- The autoimmunity-associated gene PTPN22 potentiates toll-like receptor-driven, type 1 interferon-dependent immunity.
- 2012 Mpls.St. Paul Magazine Top Doctor
- 2009-2010 Best Doctors in America
- Autoimmune Disorders, Systemic Lupus Erythematosus
- Member Minnesota Medical Association/Twin Cities
- Children'S Hosp, Pediatrics
- Children's Hospital
- Boston Combined Residency Program in Pediatrics 1999
- Children's Hospital Boston (Pediatric Rheumatology) Pediatric
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