Dr. Gregory Alan Brent MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
11301 Wilshire Blvd Endocrinology And Di Los Angeles CA, 90073Rating
1/5
About
Dr. Gregory Brent practices Endocrinology in Los Angeles, CA. Dr. Brent specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Brent examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Univ of Southern Ca Sch of Med, Los Angeles Ca 1981
Keck School of Medicine of the University of Southern California 1981
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Thyroid hormone action: down novel paths. Focus on "thyroid hormone induces activation of mitogen-activated protein kinase in cultured cells".
- Maternal hypothyroidism: recognition and management.
- Retinoic acid induces sodium/iodide symporter gene expression and radioiodide uptake in the MCF-7 breast cancer cell line.
- Induction of follicle formation in long-term cultured normal human thyroid cells treated with thyrotropin stimulates iodide uptake but not sodium/iodide symporter messenger RNA and protein expression.
- Thyroid hormone--sympathetic interaction and adaptive thermogenesis are thyroid hormone receptor isoform--specific.
- Differential regulation of the human sodium/iodide symporter gene promoter in papillary thyroid carcinoma cell lines and normal thyroid cells.
- Thyroid hormone stimulation of osteocalcin gene expression in ROS 17/2.8 cells is mediated by transcriptional and post-transcriptional mechanisms.
- Tissue-specific actions of thyroid hormone: insights from animal models.
- Nonthyroidal illness syndrome: evaluation of thyroid function in sick patients.
- A targeted thyroid hormone receptor alpha gene dominant-negative mutation (P398H) selectively impairs gene expression in differentiated embryonic stem cells.
- Thyroid hormone gene targets in ROS 17/2.8 osteoblast-like cells identified by differential display analysis.
- A thyroid-specific far-upstream enhancer in the human sodium/iodide symporter gene requires Pax-8 binding and cyclic adenosine 3',5'-monophosphate response element-like sequence binding proteins for full activity and is differentially regulated in no
- A complex deoxyribonucleic acid response element in the rat Ca(2+)/calmodulin-dependent protein kinase IV gene 5'-flanking region mediates thyroid hormone induction and chicken ovalbumin upstream promoter transcription factor 1 repression.
- The modified firefly luciferase reporter gene (luc+) but not Renilla luciferase is induced by all-trans retinoic acid in MCF-7 breast cancer cells.
- A thyroid hormone receptor alpha gene mutation (P398H) is associated with visceral adiposity and impaired catecholamine-stimulated lipolysis in mice.
Treatments
- Diabetes
- Type 2 Diabetes
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RONALD REAGAN U C L A MEDICAL CENTERl
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1250 16TH STREET SANTA MONICA CA 90404