Dr. Ralph E Whatley MD
Pulmonologist | Pulmonary Disease
1906 Belleview Ave SE Roanoke VA, 24014About
Dr. Ralph Whatley practices Pulmonology in Roanoke, VA. A pulmonologist is a physician who possesses specialized knowledge and skill in the diagnosis and treatment of pulmonary conditions and diseases. Dr. Whatley manages patients who need life support and mechanical ventilation, and is specially trained in diseases and conditions of the chest, particularly pneumonia, asthma, tuberculosis, emphysema, and complicated chest infections.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Regulation of platelet-activating factor (PAF) synthesis and PAF-mediated neutrophil adhesion to endothelial cells activated by thrombin.
- Activation of the acetyl-coenzyme A:lysoplatelet-activating factor acetyltransferase regulates platelet-activating factor synthesis in human endothelial cells.
- Relative amounts of 1-O-alkyl- and 1-acyl-2-acetyl-sn-glycero-3-phosphocholine in stimulated endothelial cells.
- Endothelial cells reestablish functional integrity after reversible permeabilization.
- Prolonged hypofibrinogenemia and protein C activation after envenoming by Echis carinatus sochureki.
- Lipid metabolism and signal transduction in endothelial cells.
- Endothelial cells for studies of platelet-activating factor and arachidonate metabolites.
- Cryobiopsy: should this be used in place of endobronchial forceps biopsies?
- The regulation of platelet-activating factor production in endothelial cells. The role of calcium and protein kinase C.
- Endothelium from diverse vascular sources synthesizes platelet-activating factor.
- Hydrogen peroxide stimulates the synthesis of platelet-activating factor by endothelium and induces endothelial cell-dependent neutrophil adhesion.
- Production of platelet-activating factor, a biologically active lipid, by vascular endothelial cells.
- The regulation of synthesis of platelet-activating factor by endothelial cells.
- Proliferation-dependent changes in release of arachidonic acid from endothelial cells.
- Growth-dependent changes in arachidonic acid release from endothelial cells are mediated by protein kinase C and changes in diacylglycerol.
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