John C Burnett M.D.
Cardiologist | Cardiovascular Disease
200 1st St SW Rochester MN, 55905About
Dr. John Burnett is a cardiologist practicing in Rochester, MN. Dr. Burnett specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Burnett also practices preventative medicine, helping patients maintain a heart-healthy life.
Education and Training
Loyola University Chicago Stritch School of Medicine 1974
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Differential actions of vasopeptidase inhibition versus angiotensin-converting enzyme inhibition on diuretic therapy in experimental congestive heart failure.
- Cardiotrophin-1 stimulation of cardiac fibroblast growth: roles for glycoprotein 130/leukemia inhibitory factor receptor and the endothelin type A receptor.
- Maximizing the natriuretic peptide system in experimental heart failure: subcutaneous brain natriuretic peptide and acute vasopeptidase inhibition.
- Adrenomedullin: an autocrine/paracrine factor for cardiorenal protection.
- The potential of brain natriuretic peptide as a biomarker for New York Heart Association class during the outpatient treatment of heart failure.
- CNP production in the kidney and effects of protein intake restriction in nephrotic syndrome.
- Cardiotrophin-1 stimulates endothelin-1 via gp130 in vascular endothelial cells.
- Plasma brain natriuretic peptide concentration: impact of age and gender.
- Natriuretic peptide receptors and neutral endopeptidase in mediating the renal actions of a new therapeutic synthetic natriuretic peptide dendroaspis natriuretic peptide.
- Brain natriuretic Peptide is produced in cardiac fibroblasts and induces matrix metalloproteinases.
- Cardiorenal and humoral properties of a novel direct soluble guanylate cyclase stimulator BAY 41-2272 in experimental congestive heart failure.
- Leukemia inhibitory factor is augmented in the heart in experimental heart failure.
- Ventricular adrenomedullin is associated with myocyte hypertrophy in human transplanted heart.
- Angiotensin II AT1 receptor antagonism prevents detrimental renal actions of acute diuretic therapy in human heart failure.
- Revisiting salt and water retention: new diuretics, aquaretics, and natriuretics.
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