Dr. Alison Rae Moliterno M.D.
Hematologist (Blood Specialist) | Hematology
1800 Orleans St Baltimore MD, 21287About
Dr. Alison Moliterno is a hematology internist practicing in Baltimore, MD. Dr. Moliterno specializes in cases of suspected blood disorders when a diagnosis is unclear and further medical care is needed. Hematology internists work with other specialists such as surgeons, radiation therapists and gynecologists in order to get a better understanding on the cases. Often, patients are asked to participate in clinical research that could result in more efficient treatment methods.
Education and Training
University At Buffalo SUNY School Of Med and Biomedical Sciences Buffalo Ny 1989
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Hematology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Posttranslational processing of the thrombopoietin receptor is impaired in polycythemia vera.
- A novel thrombopoietin signaling defect in polycythemia vera platelets.
- Biology and management of idiopathic myelofibrosis.
- Mpl Baltimore: a thrombopoietin receptor polymorphism associated with thrombocytosis.
- Wasted sheep and premature infants: the role of trace metals in hematopoiesis.
- Thrombotic and bleeding complications in four subpopulations of patients with essential thrombocythemia defined by c-Mpl protein expression and PRV-1 mRNA levels.
- Platelet counts differ by sex, ethnicity, and age in the United States.
- Molecular mimicry in the chronic myeloproliferative disorders: reciprocity between quantitative JAK2 V617F and Mpl expression.
- An absorbing problem.
- The quantitative JAK2 V617F neutrophil allele burden does not correlate with thrombotic risk in essential thrombocytosis.
- Hypersplenism induced by splenic vein ligation.
- Phenotypic variations and new mutations in JAK2 V617F-negative polycythemia vera, erythrocytosis, and idiopathic myelofibrosis.
- Phenotypic variability within the JAK2 V617F-positive MPD: roles of progenitor cell and neutrophil allele burdens.
- Human-induced pluripotent stem cells from blood cells of healthy donors and patients with acquired blood disorders.
- Acute cardiomyopathy in a patient with thrombotic thrombocytopenic purpura.
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