Dr. James L m Ferrara MD
Hematologist (Pediatric) | Pediatric Hematology-Oncology
1500 East Medical Center Dr 7th Floor Cs Mott Ch Ann Arbor MI, 48109About
Dr. James Ferrara is a pediatric hematologist practicing in Ann Arbor, MI. Dr. Ferrara specializes in treating children that have a blood disease or cancer. Such blood diseases include disorders of red blood cells, white blood cells and/or platelets. The types of cancers that Dr. Ferrara treats include leukemias, lymphomas and certain tumors. Dr. Ferrara can also treat bleeding disorders in children. Pediatric hematologists can be found in childrens hospitals, community hospitals, university medical centers and more.
Education and Training
Georgetown Univ Sch of Med, Washington Dc 1980
Board Certification
PediatricsAmerican Board of PediatricsABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Cellular and molecular mechanisms of graft-versus-host disease.
- Prevention of transfusion-associated graft-versus-host disease by photochemical treatment.
- Granulocyte colony-stimulating factor-mobilized allogeneic stem cell transplantation maintains graft-versus-leukemia effects through a perforin-dependent pathway while preventing graft-versus-host disease.
- Pretransplant chemotherapy reduces inflammatory cytokine production and acute graft-versus-host disease after allogeneic bone marrow transplantation.
- Keratinocyte growth factor separates graft-versus-leukemia effects from graft-versus-host disease.
- IL-11 separates graft-versus-leukemia effects from graft-versus-host disease after bone marrow transplantation.
- Differential roles of IL-1 and TNF-alpha on graft-versus-host disease and graft versus leukemia.
- The p55 TNF-alpha receptor plays a critical role in T cell alloreactivity.
- G-CSF modulates cytokine profile of dendritic cells and decreases acute
- Cytokines and the regulation of tolerance.
- The primacy of the gastrointestinal tract as a target organ of acute graft-versus-host disease: rationale for the use of cytokine shields in allogeneic bone marrow transplantation.
- Monoclonal antibody and receptor antagonist therapy for GVHD.
- Pathogenesis of acute graft-versus-host disease: cytokines and cellular effectors.
- Tumor necrosis factor-alpha neutralization reduces lung injury after experimental allogeneic bone marrow transplantation.
- Hyporesponsiveness of donor cells to lipopolysaccharide stimulation reduces the severity of experimental idiopathic pneumonia syndrome: potential role for a gut-lung axis of inflammation.
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