I have been practicing as a solo orthopedic surgeon since 1981. I have observed and used many different techniques for doing surgery over the years, and find that there are some that still remain that give the best long term outcomes. I have concentrated on arthroscopic surgeries of the knee primarily and enjoy an interest in spine surgery. I still concentrate on these two areas .
Education and Training
Oschner Foundation and Clinic , New Orleans, Lousiana post-docstorate 1980
In Univ Sch of Med, Indianapolis In 1974
Orthopaedic SurgeryAmerican Board of Orthopaedic SurgeryABOS
Dr. Ian J. Reynolds MD's Expert Contributions
Very possible! READ MORE
It sounds more like there is a problem with your rotator cuff tendon READ MORE
Muscles are pulling in their attachments to the bone, need to slow down a bit and see if it abates. READ MORE
You need to see an orthopedic sports medicine doctor READ MORE
Yes, it can progress. READ MORE
Do what Mother Nature will let you do without pain! READ MORE
It is worth a try. The amount given will not cause any side effects! READ MORE
Not usually. Most often, it can be relieved with cortisone injections. READ MORE
He needs to get an MRI of his neck and shoulders to look for the cause. READ MORE
It depends on the joint: if it is the hip, it can be replaced; if it is a small area in the knee, it may be tolerable, but replacement may also be needed. READ MORE
in most cases it will due to the lack of microcirculation READ MORE
Muscle spasm - probably from a strained facet joint! Try an anti-inflammatory, such as ibuprofen, heat, and a massage. READ MORE
Just routine rest, ice, compress, elevate! READ MORE
No, it is not true, it will not weaken the bones, just creating negative pressure in the joint and it makes a pop! READ MORE
At age 40 - just make sure you eat green, leafy vegetables for the calcium and add vitamin D3 over the counter, check Vit D levels to see if you need D2. READ MORE
First, get an X-ray to see if there are spurs, change your shoes every three days, wear heel cups with a hole in the center, take anti-inflammatories, maybe get a cortisone injection, and, lastly, surgery to remove the spur. READ MORE
Most likely it is, but not proven! READ MORE
Keeps coming back, but can be controlled with daily medicine. READ MORE
I am not that familiar with ankle arthroplasty, my experience is that they do not work well in very active individuals, however swimming would be the best rehab exercise I could think of for this procedure READ MORE
No need for knee replacement if there is no pain just noise ! READ MORE
In most cases people are back to full speed in 6 weeks READ MORE
Most likely you have an area of soft cartilage on the back of your knee cap that is periodically irritating , it can result in severe pain when bending your knee and be bothersome at times at night , if it persists despite taking an aspirin for several weeks per day the you need to get at an MRI READ MORE
It could be that the extra effort in walking is taxing the medial hamstrings or adductor tendons a little more , so this is either a tendinitis issue or it may be a little nerve root irritation related to S1 root (1st sacral) , first try stretching the inner thighs and see if it makes a difference READ MORE
It depends on what area you are calling your hip , if it is ne'er the buttock it might be coming from your back , if it is in the groin area it could be the hip joint itself or a pulled muscle such as rectus femoris . If rest and a non steroidal doesn't clear it up in a few weeks , I would suggest an X-ray of the hip to start READ MORE
Repairing an ACL tear will not work , the only way to stabilize the knee is to replace the ACL with a new tendon graft ! READ MORE
- High-affinity calcium indicators underestimate increases in intracellular calcium concentrations associated with excitotoxic glutamate stimulations.
- Glutamate-induced neuron death requires mitochondrial calcium uptake.
- Characterization of the effects of polyamines on [125I]MK-801 binding to recombinant N-methyl-D-aspartate receptors.
- The use of ligand binding in assays of NMDA receptor function.
- Novel bisbenzamidines and bisbenzimidazolines as noncompetitive NMDA receptor antagonists.
- Emergence of excitotoxicity in cultured forebrain neurons coincides with larger glutamate-stimulated [Ca(2+)](i) increases and NMDA receptor mRNA levels.
- Mitochondrial membrane potential and the permeability transition in excitotoxicity.
- Subcellular localization of glutamate-stimulated intracellular magnesium concentration changes in cultured rat forebrain neurons using confocal microscopy.
- Inhibition of glutamate-induced mitochondrial depolarization by tamoxifen in cultured neurons.
- Astrocytes are more resistant than neurons to the cytotoxic effects of increased [Zn(2+)](i).
- Induction of neuronal apoptosis by thiol oxidation: putative role of intracellular zinc release.
- Pharmacological investigation of mitochondrial ca(2+) transport in central neurons: studies with CGP-37157, an inhibitor of the mitochondrial Na(+)-Ca(2+) exchanger.
- MitoTracker labeling in primary neuronal and astrocytic cultures: influence of mitochondrial membrane potential and oxidants.
- Quantitative evaluation of mitochondrial calcium content in rat cortical neurones following a glutamate stimulus.
- Spontaneous changes in mitochondrial membrane potential in cultured neurons.
- Vanilloid receptor expression suggests a sensory role for urinary bladder epithelial cells.
- DeltaPsi(m)-Dependent and -independent production of reactive oxygen species by rat brain mitochondria.
- Nitric oxide-induced changes in intracellular zinc homeostasis are mediated by metallothionein/thionein.
- The relationship between intracellular free iron and cell injury in cultured neurons, astrocytes, and oligodendrocytes.
- Nitric oxide decreases the sensitivity of pulmonary endothelial cells to LPS-induced apoptosis in a zinc-dependent fashion.
- A reevaluation of neuronal zinc measurements: artifacts associated with high intracellular dye concentration.
- Elevated intracellular zinc and altered proton homeostasis in forebrain neurons.
- Zinc inhibition of cellular energy production: implications for mitochondria and neurodegeneration.
- New perspectives on mitochondrial morphology in cell function.
- Glutamate decreases mitochondrial size and movement in primary forebrain neurons.
- Synthesis and biological activity of 8a-phenyldecahydroquinolines as probes of PCP's binding conformation. A new PCP-like compound with increased in vivo potency.
- [125I]thienylphencyclidine, a novel ligand for the NMDA receptor.
- Synthesis and evaluation of fluoromycin: a novel fluorescence-labeled derivative of talisomycin S10b.
- Interactions between zinc and spermidine on the N-methyl-D-aspartate receptor complex: clues to the mechanism of action of 1,10-bis(guanidino)decane and pentamidine.
- Nitric oxide modulates NMDA-induced increases in intracellular Ca2+ in cultured rat forebrain neurons.
- Glucose deprivation produces a prolonged increase in sensitivity to glutamate in cultured rat cortical neurons.
- Spontaneous changes in mitochondrial membrane potential in single isolated brain mitochondria.
- Ectopic expression of the catalytic subunit of telomerase protects against brain injury resulting from ischemia and NMDA-induced neurotoxicity.
- Divergent consequences arise from metallothionein overexpression in astrocytes: zinc buffering and oxidant-induced zinc release.
- Epidermal growth factor activates m-calpain (calpain II), at least in part, by extracellular signal-regulated kinase-mediated phosphorylation.
- Fluorescence detection of redox-sensitive metals in neuronal culture: focus on iron and zinc.
- Fluctuations in mitochondrial membrane potential in single isolated brain mitochondria: modulation by adenine nucleotides and Ca2+.
- Pentamidine is an N-methyl-D-aspartate receptor antagonist and is neuroprotective in vitro.
- Effects of monovalent and divalent cations on 3-(+)[125I]iododizocilpine binding to the N-methyl-D-aspartate receptor of rat brain membranes.
- 1,5-(Diethylamino)piperidine, a novel spermidine analogue that more specifically activates the N-methyl-D-aspartate receptor-associated polyamine site.
- Mitochondrial trafficking in neurons: a key variable in neurodegeneration?
- Mitochondrial stop and go: signals that regulate organelle movement.
- Detection of hydrogen peroxide with Amplex Red: interference by NADH and reduced glutathione auto-oxidation.
- Simultaneous detection of intracellular free calcium and zinc using fura-2FF and FluoZin-3.
- Synaptosomal dopamine uptake in rat striatum following controlled cortical impact.
- Ca2+-induced permeabilization promotes free radical release from rat brain mitochondria with partially inhibited complex I.
- Direct visualization of mitochondrial zinc accumulation reveals
- Dopaminergic neurotoxins require excitotoxic stimulation in organotypic cultures.
- Zinc causes loss of membrane potential and elevates reactive oxygen species in rat brain mitochondria.
- Distinct characteristics of Ca(2+)-induced depolarization of isolated brain and liver mitochondria.
- Zn2+ inhibits mitochondrial movement in neurons by phosphatidylinositol 3-kinase activation.
- Modulation of NMDA excitotoxicity by redox reagents.
- Mutant huntingtin aggregates impair mitochondrial movement and trafficking in cortical neurons.
- Nitric oxide inhibits mitochondrial movement in forebrain neurons associated with disruption of mitochondrial membrane potential.
- 1,10-bis(guanidino)decane inhibits N-methyl-D-aspartate responses in vitro and in vivo.
- Differences in mitochondrial movement and morphology in young and mature primary cortical neurons in culture.
- Mitochondrial trafficking to synapses in cultured primary cortical neurons.
- Mitochondrial trafficking and morphology in healthy and injured neurons.
- Nuclear and mitochondrial interaction involving mt-Nd2 leads to increased mitochondrial reactive oxygen species production.
- Regional variations in [3H]MK801 binding to rat brain N-methyl-D-aspartate receptors.
- The spider toxin, argiotoxin636, binds to a Mg2+ site on the N-methyl-D-aspartate receptor complex.
- Effects of nicotinic agonists on the NMDA receptor.
- Effects of age and visual experience on [3H] MK801 binding to NMDA receptors in the kitten visual cortex.
- Thermal nociception and TRPV1 function are attenuated in mice lacking the nucleotide receptor P2Y2.
- Oxidized glutathione modulates N-methyl-D-aspartate- and depolarization-induced increases in intracellular Ca2+ in cultured rat forebrain neurons.
- Measurement of cation movement in primary cultures using fluorescent dyes.
- Glutamate mobilizes [Zn2+] through Ca2+ -dependent reactive oxygen species accumulation.
- Discovery of 1,4-substituted piperidines as potent and selective inhibitors of T-type calcium channels.
- Mitochondrial trafficking and morphology in neuronal injury.
- Modulation of NMDA receptor responsiveness by neurotransmitters, drugs and chemical modification.
- PISA, a novel pharmacodynamic assay for assessing poly(ADP-ribose) polymerase (PARP) activity in situ.
- Apolipoprotein E4 domain interaction mediates detrimental effects on mitochondria and is a potential therapeutic target for Alzheimer disease.
- Arcaine uncovers dual interactions of polyamines with the N-methyl-D-aspartate receptor.
- Allosteric modulation of N-methyl-D-aspartate receptors.
- High throughput monitoring of amyloid-β(42) assembly into soluble oligomers achieved by sensitive conformation state-dependent immunoassays.
- Oxygen free radicals regulate NMDA receptor function via a redox modulatory site.
- Arcaine is a competitive antagonist of the polyamine site on the NMDA receptor.
- Synthesis and bioactivity of a new class of rigid glutamate analogues. Modulators of the N-methyl-D-aspartate receptor.
- Attenuation of scratch-induced reactive astrogliosis by novel EphA4 kinase inhibitors.
- [3H](+)MK801 radioligand binding assay at the N-methyl-D-aspartate receptor.
- A potent and selective metabotropic glutamate receptor 4 positive allosteric modulator improves movement in rodent models of Parkinson's disease.
- Calcium-antagonist drugs. Receptor interactions that clarify therapeutic effects.
- Calcium antagonist receptors. Clinical and physiological relevance.
- Physiological and pharmacological correlates of calcium antagonist receptors.
- Characterization of the novel positive allosteric modulator of the metabotropic glutamate receptor 4 ADX88178 in rodent models of neuropsychiatric disorders.
- NMDA receptor antagonists that bind to the strychnine-insensitive glycine site and inhibit NMDA-induced Ca2+ fluxes and [3H]GABA release.
- Alterations in calcium antagonist receptors and sodium-calcium exchange in cardiomyopathic hamster tissues.
- Guanine nucleotides are competitive inhibitors of N-methyl-D-aspartate at its receptor site both in vitro and in vivo.
- Ifenprodil is a novel type of N-methyl-D-aspartate receptor antagonist: interaction with polyamines.
- Muscarinic agonists cause calcium influx and calcium mobilization in forebrain neurons in vitro.
- 3H-labeled MK-801 binding to the excitatory amino acid receptor complex from rat brain is enhanced by glycine.
- Inhibition of the mitochondrial pyruvate carrier protects from excitotoxic neuronal death.
- Drug repurposing from the perspective of pharmaceutical companies.
- Multiple sites for the regulation of the N-methyl-D-aspartate receptor.
- [3H]MK801 binding to the NMDA receptor/ionophore complex is regulated by divalent cations: evidence for multiple regulatory sites.
- [3H]MK801 binding to the N-methyl-D-aspartate receptor reveals drug interactions with the zinc and magnesium binding sites.
- Calcium antagonist receptors.
- Tricyclic antidepressants block N-methyl-D-aspartate receptors: similarities to the action of zinc.
- Calcium antagonist receptors in cardiomyopathic hamster: selective increases in heart, muscle, brain.
- (-)-[3H] desmethoxyverapamil labels multiple calcium channel modulator receptors in brain and skeletal muscle membranes: differentiation by temperature and dihydropyridines.
- Assay of partially neutralized guinea pig anti-insulin serum.
- Antischizophrenic drugs of the diphenylbutylpiperidine type act as calcium channel antagonists.
- [3H]Verapamil binding sites in brain and skeletal muscle: regulation by calcium.
- Calcium channel blockade: possible explanation for thioridazine's peripheral side effects.
- Loperamide: blockade of calcium channels as a mechanism for antidiarrheal effects.
- Calcium influx but not pH or ATP level mediates glutamate-induced changes in intracellular magnesium in cortical neurons.
- Cyclothiazide modulates AMPA receptor-mediated increases in intracellular free Ca2+ and Mg2+ in cultured neurons from rat brain.
- Orally administered progesterone enhances sensitivity to triazolam in postmenopausal women.
- Glutamate induces the production of reactive oxygen species in cultured forebrain neurons following NMDA receptor activation.
- Mitochondria and Na+/Ca2+ exchange buffer glutamate-induced calcium loads in cultured cortical neurons.
- Glutamate-induced intracellular calcium changes and neurotoxicity in cortical neurons in vitro: effect of chemical ischemia.
- [3H]CGP 39653 binding to the agonist site of the N-methyl-D-aspartate receptor is modulated by Mg2+ and polyamines independently of the arcaine-sensitive polyamine site.
- Calcium green-5N, a novel fluorescent probe for monitoring high intracellular free Ca2+ concentrations associated with glutamate excitotoxicity in cultured rat brain neurons.
- Glutamate-induced increases in intracellular free Mg2+ in cultured cortical neurons.
- Studies on the effects of several pentamidine analogues on the NMDA receptor.
- Effects of pH on the actions of dizocilpine at the N-methyl-D-aspartate receptor complex.
- Modulation of [3H]flunitrazepam binding by natural and synthetic progestational agents.
- Characterization of the effects of polyamines on the modulation of the N-methyl-D-aspartate receptor by glycine.
- Effect of neuroactive steroids on [3H]flumazenil binding to the GABAA receptor complex in vitro.
- Mechanisms of glutamate-stimulated Mg2+ influx and subsequent Mg2+ efflux in rat forebrain neurones in culture.
- Desensitization of 5HT2 receptors by protein kinase C activation in distal pulmonary vascular smooth muscle cells in culture.
- Mitochondrial depolarization in glutamate-stimulated neurons: an early signal specific to excitotoxin exposure.
- Localization of D1 dopamine receptors on live cultured striatal neurons by quantitative fluorescence microscopy.
- Mitochondria accumulate Ca2+ following intense glutamate stimulation of cultured rat forebrain neurones.
- Characterization of hydrogen peroxide toxicity in cultured rat forebrain neurons.
- Mechanisms of dopamine-induced cell death in cultured rat forebrain neurons: interactions with and differences from glutamate-induced cell death.
- NMDA receptor-mediated neurotoxicity: a paradoxical requirement for extracellular Mg2+ in Na+/Ca2+-free solutions in rat cortical neurons in vitro.
- Effects of pyrroloquinoline quinone on glutamate-induced production of reactive oxygen species in neurons.
- Alprazolam in young and elderly men: sensitivity and tolerance to psychomotor, sedative and memory effects.
- Trifluoperazine and dibucaine-induced inhibition of glutamate-induced mitochondrial depolarization in rat cultured forebrain neurones.
- Reverse Na+/Ca2+ exchange contributes to glutamate-induced intracellular Ca2+ concentration increases in cultured rat forebrain neurons.
- The role of intracellular Na+ and mitochondria in buffering of kainate-induced intracellular free Ca2+ changes in rat forebrain neurones.
- Alkalinization prolongs recovery from glutamate-induced increases in intracellular Ca2+ concentration by enhancing Ca2+ efflux through the mitochondrial Na+/Ca2+ exchanger in cultured rat forebrain neurons.
- Complex polyamine effects on [3H]MDL 105,519 binding to the NMDA receptor glycine site.
- Effects of oxidants and glutamate receptor activation on mitochondrial membrane potential in rat forebrain neurons.
- Calcium-sensitive fluorescent dyes can report increases in intracellular free zinc concentration in cultured forebrain neurons.
- Synthesis of a potent wide-spectrum serotonin-, norepinephrine-, dopamine-reuptake inhibitor (SNDRI) and a species-selective dopamine-reuptake inhibitor based on the gamma-amino alcohol functional group.
- Intracellular calcium and magnesium: critical determinants of excitotoxicity?
- Aromatic analogs of arcaine inhibit MK-801 binding to the NMDA receptor.
- Alton Ochsner Med Fndn, Orthopedic Surgery; Clarian Methodist Hosp Of Indi, General Surgery
- Ochsner Fdn 1975
- Orthopedics at Ochsner Health System
Faculty Titles & Positions
- Alton Ochsner Med Fndn, Orthopedic Surgery; Clarian Methodist Hosp Of Indi, General Surgery
- Ochsner Fdn 1975
Professional Society Memberships
- Texas Orthopaedic Association, Southern Orthopaedic Association, Texas Society of Sports Medicine, Southern Medical Association
What do you attribute your success to?
- Motivation that comes from seeing patients resume their functional lives after surgery and recovery.
Hobbies / Sports
- Traveling, Skiing, Various Science Projects, Water Purification Systems
Favorite professional publications
- The Journal of Bone and Joint Surgery
- Bay Area Regional , Webster Texas 77598
- Surgical Specialty Hospital , Pasadena , Texas
- Houston Hospital for Specialized Surgery ( Houston, TX )
- Aetna Advantage Dental
- Aetna Affordable Health Choices limited benefits insurance plan
- Aetna Choice POS
- Aetna Choice POS (Aetna HealthFund)
- Aetna Choice POS II
- Aetna Choice POS II (Aetna HealthFund)
- Aetna Dental PPO/PDN
- Aetna Golden Choice Plan (PPO)
- Aetna Golden Medicare Plan (HMO)
- Aetna Health Network Only
- Aetna Health Network Option
- Aetna HealthFund Aetna Choice POS
- Aetna HealthFund Aetna Health Network Only
- Aetna HealthFund Aetna Health Network Option
- Aetna HealthFund HMO
- Aetna HealthFund Open Access Aetna Select
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Dr. Ian J. Reynolds MD's Practice location
Friendswood, TX 77546Get Direction
Webster, TX 77598Get Direction
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Dr. Ian J. Reynolds MD's reviewsWrite Review
Patient Experience with Dr. Reynolds
Love the staff and Doctor! Very accommodating and professional. I would recommend to anyone!
I have had to have 4 surgeries due to various injuries. Dr. Reynolds has been excellent in explaining the conditions and remedies. He has put me back together each time. His staff is very nice and they do not mess you around like other Dr. Offices do. Very good, you will definitely appreciate the difference. Do yourself a favor choose Dr. Reynolds
Wonderful staff and service! Dr. Reynolds and office is attentive and efficient. Truly cares for patient's needs!
Dr. Reynolds has very good beside manners and he is very tolerable in listening to his patients. His staff is great in accommodating me as a patient for answering questions and being knowledgeable about places to go to and do therapy, pick up good back braces and etc.... Diane his office manager will also call you back after hours that is exceptionally well.
Great office staff !! Very friendly doc
What an awesome dr. The office staff is the best I have ever dealt with,,thanks for fixing me..I would recommend everyone to come here for all orthopedic needs..thanks again.
Get to know Orthopedic Surgeon Dr. Ian J. Reynolds, who serves the population of Webster, Texas.
Dr. Ian J. Reynolds is an established Orthopedic Surgeon currently serving patients within his own private practice in Webster, Texas. He is also affiliated with the Surgery Specialty Hospitals of America, and has privileges at Bay Area Regional Medical Center and The Hospital for Specialized Surgery in Houston. Dr. Reynolds features nearly four decades of experience in his field, and has a special expertise in sports medicine, arthroscopic surgery, joint replacement, spine surgery, ligament surgery, and surgery of the hand and foot.
Dr. Reynolds attended Indiana University School of Medicine in Indianapolis, graduating with his Medical Degree in 1971. Following the completion of medical school, he completed an internship at Methodist Hospital, prior to his residency training at the Ochsner Foundation Hospital of New Orleans. Dr. Reynolds is certified by the American Board of Orthopaedic Surgery, and has earned the coveted title of Fellow of the American Academy of Orthopaedic Surgeons.
To keep up to date with the latest advances in his field, Dr. Reynolds maintains a professional membership with the Texas Orthopaedic Association, the Southern Orthopaedic Association, the Texas Society of Sports Medicine, the Southern Medical Association, and the Texas Medical Association. He attributes his success to his motivation to help patients regain their health and mobility, and in his free time, Dr. Reynolds enjoys traveling and skiing, and he is also an amateur scientist with a special interest in water purification systems. Dr. Reynolds also has skin care anti-aging products distributed under the name JPaul Skin Care. He is a patent holder for a method of manufacturing timed released amino acids and glutamine, creatine and whey supplements, and is also interested in aviation and current has two designs for a commuter jet and a supersonic business jet that are in development.
Orthopaedic surgeons are devoted to the prevention, diagnosis, and treatment of disorders of the bones, joints, ligaments, tendons and muscles. Some orthopaedists are generalists, while others specialize in certain areas of the body, others specialty focuses include shoulder and elbow, hand, and spine. Orthopaedic surgeons treat broken bones, and replace painful joints, and also treat patients for sports injuries, back pain, ruptured discs and spinal stenosis, bone tumors, carpal tunnel, hand arthritis and hand injuries, club foot, bow legs and hip dysplasia, orthopaedic trauma, limb lengthening, achilles tendon injuries, bunions and foot and ankle injuries, osteoporosis, and arthritis.
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