Dr. Amber Thurston PT
Physical Therapist (Pediatric) | Pediatrics
6301 GASTON AVE STE 750 DALLAS TX, 75214About
Amber Thurston is a pediatric physical therapist practicing in DALLAS, TX. Amber is a doctor specializing in physical treatment of children to help reduce pain, restore mobility, rehabilitate an injury, or increase movement and overall function. As a physical therapist, Dr. Thurston can treat multiple conditions with exercises, ultrasound, electrical stimulation, joint mobilization, heat, ice, massage, laser or light therapy and more. Dr. Thurston will create a treatment plan based on the childs specific injury, disease or condition, and might target a specific body part or body system based on the individual.
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Expert Publications
Data provided by the National Library of Medicine- Acoustic realignment of nematic liquid crystals.
- Comparison of the characteristics of the chiral analog of the de Vries type of smectic-A* phase.
- Orderly dismantling of a cell.
- Theory of the acoustic realignment of nematic liquid crystals.
- Generation and analysis of genetically defined liver carcinomas derived from bipotential liver progenitors.
- Mast-cell leukemia exome sequencing reveals a mutation in the IgE mast-cell receptor β chain and KIT V654A.
- The self-association of human apolipoprotein A-IV. Evidence for an in vivo circulating dimeric form.
- Lipoprotein affinity of human apolipoprotein A-IV during cholesterol esterification.
- Human apolipoprotein A-IV: displacement from the surface of triglyceride-rich particles by HDL2-associated C-apoproteins.
- Structural properties and lipid binding of human apolipoprotein A-IV.
- Isoform heterogeneity and lipid affinity of human lymph and plasma apolipoprotein A-IV.
- The rat alpha 1B adrenergic receptor gene middle promoter contains multiple binding sites for sequence-specific proteins including a novel ubiquitous transcription factor.
- The HIR4-1 mutation defines a new class of histone regulatory genes in Saccharomyces cerevisiae.
- Interaction between the C. elegans cell-death regulators CED-9 and CED-4.
- Sp1-mediated transcriptional activation from the dominant promoter of the rat alpha1B adrenergic receptor gene in DDT1MF-2 cells.
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