Dr. Michael R Wessels MD
Infectious Disease Specialist | Infectious Disease
300 Longwood Ave Boston MA, 02115About
Dr. Michael Wessels is an infectious disease specialist practicing in Boston, MA. Dr. Wessels specializes in infections that are difficult to diagnose or unresponsive to treatments, such as HIV or airborne infections from a foreign country. Infectious disease specialists usually work with conditions that are not treatable by a primary physician but it is important to keep contact with the primary physician in order to receive information about the patients history and for deciding which diagnostic tests are appropriate.
Education and Training
Duke University School of Medicine 1979
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Infectious Disease
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- NAD+-glycohydrolase acts as an intracellular toxin to enhance the extracellular survival of group A streptococci.
- The CsrR/CsrS two-component system of group A Streptococcus responds to environmental Mg2+.
- Critical role of the complement system in group B streptococcus-induced tumor necrosis factor alpha release.
- Selective impairment of TLR-mediated innate immunity in human newborns: neonatal blood plasma reduces monocyte TNF-alpha induction by bacterial lipopeptides, lipopolysaccharide, and imiquimod, but preserves the response to R-848.
- Interaction between complement regulators and Streptococcus pyogenes: binding of C4b-binding protein and factor H/factor H-like protein 1 to M18 strains involves two different cell surface molecules.
- Regulation of virulence by a two-component system in group B streptococcus.
- Cytolysin-dependent evasion of lysosomal killing.
- Streptolysin S.
- Extracellular group A Streptococcus induces keratinocyte apoptosis by dysregulating calcium signalling.
- Genome analysis of multiple pathogenic isolates of Streptococcus agalactiae: implications for the microbial "pan-genome".
- Role of NADase in virulence in experimental invasive group A streptococcal infection.
- Changing epidemiology of acute rheumatic fever in the United States.
- Enhancement of streptolysin O activity and intrinsic cytotoxic effects of the group A streptococcal toxin, NAD-glycohydrolase.
- Unique efficacy of Toll-like receptor 8 agonists in activating human neonatal antigen-presenting cells.
- The adenosine system selectively inhibits TLR-mediated TNF-alpha production in the human newborn.
Awards
- 2011 Boston Super Doctors
Fellowships
- Channing Laboratory, Brigham & Women's Hospital - Boston, MA
- Brigham & Womens/Beth Israel, Fellow:Infectious Diseases 1982
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