Dr. George Arthur Porter MD
Cardiologist (Pediatric) | Pediatric Cardiology
20 York St Ynhh Children's Hosp New Haven CT, 06510About
Dr. George Porter is a pediatric cardiologist practicing in New Haven, CT. Dr. Porter specializes in caring for fetuses, infants, children and adolescents with cardiovascular or cardiac abnormalities. Practiced in both inpatient and outpatient settings, the scope of conditions cared for by pediatric cardiologists is large and includes congenital heart defects, heart muscle disorders, rhythm disturbances and hypertension.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- The medical consequences of nuclear war: introduction.
- Sinus node dysfunction associated with lithium therapy in a child.
- Non-contrast-enhanced computerized tomography and analgesic-related kidney disease: report of the national analgesic nephropathy study.
- Novel sorbents for removal of gadolinium-based contrast agents in sorbent dialysis and hemoperfusion: preventive approaches to nephrogenic systemic fibrosis.
- Bioenergetics, mitochondria, and cardiac myocyte differentiation.
- The permeability transition pore controls cardiac mitochondrial maturation and myocyte differentiation.
- Mitochondria as a drug target in ischemic heart disease and cardiomyopathy.
- SIRT3 deficiency exacerbates ischemia-reperfusion injury: implication for aged hearts.
- Bcl-xL in neuroprotection and plasticity.
- Initiation of electron transport chain activity in the embryonic heart coincides with the activation of mitochondrial complex 1 and the formation of supercomplexes.
- Cell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthase.
- Extraembryonic but not embryonic SUMO-specific protease 2 is required for heart development.
- The Mitochondrial Permeability Transition Pore and ATP Synthase.
- Erratum to: The Mitochondrial Permeability Transition Pore and ATP Synthase.
- Preventing permeability transition pore opening increases mitochondrial maturation, myocyte differentiation and cardiac function in the neonatal mouse heart.
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