Dr. Joseph A Lorenzo MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
263 Farmington Ave Farmington CT, 06030About
Dr. Lorenzo is a professor of medicine in the Division of Endocrinology and Metabolism at the UConn Health Center. He is a nationally renowned expert in endocrinology and metabolic bone diseases. Dr. ...
Education and Training
Suny-Hlth Sci Ctr At Brooklyn, Coll of Med, Brooklyn Ny 1974
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- 1,25(OH)2 vitamin D3-stimulated osteoclast formation in spleen-osteoblast cocultures is mediated in part by enhanced IL-1 alpha and receptor activator of NF-kappa B ligand production in osteoblasts.
- Increased bone mass is a part of the generalized lymphoproliferative disorder phenotype in the mouse.
- Interleukin-7 is a direct inhibitor of in vitro osteoclastogenesis.
- Megakaryocyte-osteoblast interaction revealed in mice deficient in transcription factors GATA-1 and NF-E2.
- Pax5-deficient mice exhibit early onset osteopenia with increased osteoclast progenitors.
- Biphasic effect of prostaglandin E2 on osteoclast formation in spleen cell cultures: role of the EP2 receptor.
- Osteoclast differentiation independent of the TRANCE-RANK-TRAF6 axis.
- RANKL-stimulated osteoclast-like cell formation in vitro is partially dependent on endogenous interleukin-1 production.
- Identification of multiple osteoclast precursor populations in murine bone marrow.
- Interleukin-7 influences osteoclast function in vivo but is not a critical factor in ovariectomy-induced bone loss.
- Megakaryocyte-mediated inhibition of osteoclast development.
- T lymphocyte-deficient mice lose trabecular bone mass with ovariectomy.
- B lymphocytes and the skeleton.
Awards
- Career Development Award, 1983-1988 VA Research Associate
- New Investigator Research Award, 1982-1985 NIH
Fellowships
- University of Connecticut School of Medicine, Endocrinology/Metabolism
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