Dr. Maria C. Jong MD
Psychiatrist | Psychiatry
1011 Baldwin Park Blvd Baldwin Park CA, 91706Rating
5/5
About
Dr. Maria Jong is a psychiatrist practicing in Baldwin Park, CA. Dr. Jong is a medical doctor specializing in the care of mental health patients. As a psychiatrist, Dr. Jong diagnoses and treats mental illnesses. Dr. Jong may treat patients through a variety of methods including medications, psychotherapy or talk therapy, psychosocial interventions and more, depending on each individual case. Different medications that a psychiatrist might prescribe include antidepressants, antipsychotic mediations, mood stabilizers, stimulants, sedatives and hypnotics. Dr. Jong treats conditions like depression, anxiety, OCD, eating disorders, bipolar disorders, personality disorders, insomnia, ADD and other mental illnesses.
Education and Training
Northwestern Uniiversity 1990
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Reversal of hyperlipidaemia in apolipoprotein C1 transgenic mice by adenovirus-mediated gene delivery of the low-density-lipoprotein receptor, but not by the very-low-density-lipoprotein receptor.
- Role of ApoCs in lipoprotein metabolism: functional differences between ApoC1, ApoC2, and ApoC3.
- Apolipoprotein E participates in the regulation of very low density lipoprotein-triglyceride secretion by the liver.
- Oxidized VLDL induces less triglyceride accumulation in J774 macrophages than native VLDL due to an impaired extracellular lipolysis.
- Cafestol increases serum cholesterol levels in apolipoprotein E*3-Leiden transgenic mice by suppression of bile acid synthesis.
- Apolipoprotein E2 (Lys146-->Gln) causes hypertriglyceridemia due to an apolipoprotein E variant-specific inhibition of lipolysis of very low density lipoproteins-triglycerides.
- Insights into apolipoprotein C metabolism from transgenic and gene-targeted mice.
- Hepatic lipid accumulation, altered very low density lipoprotein formation and apolipoprotein E deposition in apolipoprotein E3-Leiden transgenic mice.
- LDL receptor deficiency unmasks altered VLDL triglyceride metabolism in VLDL receptor transgenic and knockout mice.
- Hyperlipidaemia is associated with increased insulin-mediated glucose metabolism, reduced fatty acid metabolism and normal blood pressure in transgenic mice overexpressing human apolipoprotein C1.
- Protection from obesity in mice lacking the VLDL receptor.
- Apolipoprotein C-III deficiency accelerates triglyceride hydrolysis by lipoprotein lipase in wild-type and apoE knockout mice.
- In muscle-specific lipoprotein lipase-overexpressing mice, muscle triglyceride content is increased without inhibition of insulin-stimulated whole-body and muscle-specific glucose uptake.
- Protection from obesity and insulin resistance in mice overexpressing human apolipoprotein C1.
Dr. Maria C. Jong MD's Practice location
Practice At 1011 Baldwin Park Blvd
1011 Baldwin Park Blvd -Baldwin Park, CA 91706Get Direction
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