Dr. Steven Hunt Graham M.D.
Neurologist | Neurology
Uninversity Drive C - V. A. Medical Center Neurology Pittsburgh PA, 15217About
Dr. Steven Graham is a distinguished Neurologist in Pittsburgh, PA. Dr. Graham specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Graham employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Graham is committed to staying abreast of the latest developments in neurological research and therapies.
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Expert Publications
Data provided by the National Library of Medicine- Induction of caspase-activated deoxyribonuclease activity after focal cerebral ischemia and reperfusion.
- Cloning and characterization of rat caspase-9: implications for a role in mediating caspase-3 activation and hippocampal cell death after transient cerebral ischemia.
- Regulation of interstitial excitatory amino acid concentrations after cortical contusion injury.
- Two caspase-2 transcripts are expressed in rat hippocampus after global cerebral ischemia.
- Regulation of interstitial excitatory amino acid concentrations after cortical contusion injury.
- In Vivo Delivery of a Bcl-xL Fusion Protein Containing the TAT Protein Transduction Domain Protects against Ischemic Brain Injury and Neuronal Apoptosis.
- Increased S-nitrosothiols and S-nitrosoalbumin in cerebrospinal fluid after severe traumatic brain injury in infants and children: indirect association with intracranial pressure.
- Induced hyperthermia exacerbates neurologic neuronal histologic damage after asphyxial cardiac arrest in rats.
- Cyclooxygenases in central nervous system diseases: a special role for cyclooxygenase 2 in neuronal cell death.
- Translocation of apoptosis-inducing factor in vulnerable neurons after transient cerebral ischemia and in neuronal cultures after oxygen-glucose deprivation.
- c-FLIP-L recombinant adeno-associated virus vector infection prevents Fas-mediated but not nerve growth factor withdrawal-mediated cell death in PC12 cells.
- Innate gender-based proclivity in response to cytotoxicity and programmed cell death pathway.
- Arachidonic acid-induced carbon-centered radicals and phospholipid peroxidation in cyclo-oxygenase-2-transfected PC12 cells.
- Cyclooxygenase-2 activity contributes to neuronal expression of cyclin D1 after anoxia/ischemia in vitro and in vivo.
- Neuroprotection against focal ischemic brain injury by inhibition of c-Jun N-terminal kinase and attenuation of the mitochondrial apoptosis-signaling pathway.
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Steven Graham
Uninversity Drive C - V. A. Medical Center Neurology -Pittsburgh, PA 15217Get Direction
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