Dr. Lee Anne Campbell D.D.S.
Dentist | General Practice
202 First Street S Yelm WA, 98597About
Dr. Lee Campbell is a Dentist practicing in Yelm, WA. Dr. Campbell specializes in preventing, diagnosing, and treating diseases and conditions associated with the mouth and overall dental health. Dentists are trained to carry out such treatment as professional cleaning, restorative, prosthodontic, and endodontic procedures, and performing examinations, among many others.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Mannose-receptor positive and negative mouse macrophages differ in their susceptibility to infection by Chlamydia species.
- Chlamydia pneumoniae infection and atherosclerosis: methodological considerations.
- Foam cell formation inhibits growth of Chlamydia pneumoniae but does not attenuate Chlamydia pneumoniae-induced secretion of proinflammatory cytokines.
- Chlamydia pneumoniae pathogenesis.
- Chlamydia pneumoniae and hyperlipidemia are co-risk factors for atherosclerosis: infection prior to induction of hyperlipidemia does not accelerate development of atherosclerotic lesions in C57BL/6J mice.
- Chlamydial infections of the cardiovascular system.
- Intrastrain and interstrain genetic variation within a paralogous gene family in Chlamydia pneumoniae.
- Cell-to-cell contact of human monocytes with infected arterial smooth-muscle cells enhances growth of Chlamydia pneumoniae.
- Chlamydia pneumoniae induces tissue factor expression in mouse macrophages via activation of Egr-1 and the MEK-ERK1/2 pathway.
- Chlamydia pneumoniae and atherosclerosis.
- Nitric oxide synthase plays a role in Chlamydia pneumoniae-induced atherosclerosis.
- Chronic inhibition of cyclooxygenase-2 does not alter plaque composition in a mouse model of advanced unstable atherosclerosis.
- Chlamydia pneumoniae--an infectious risk factor for atherosclerosis?
- Amphiphysin IIm is required for survival of Chlamydia pneumoniae in macrophages.
- Lesion progression and plaque composition are not altered in older apoE-/- mice lacking tumor necrosis factor-alpha receptor p55.
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