Dr. Eric L Klett MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
300 Meadowmont Village Circle Suite 202 Chapel Hill NC, 27517About
Dr. Eric Klett practices Endocrinology in West Columbia, SC. Dr. Klett specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Klett examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Med Univ of Sc Coll of Med, Charleston Sc 1999
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Endocrinology, Diabetes and Metabolism
Internal MedicineAmerican Board of Internal MedicineABIM- 2003
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Genetic defenses against noncholesterol sterols.
- Biomedicine. Will the real cholesterol transporter please stand up.
- Hepatic triacylglycerol accumulation and insulin resistance.
- Acyl-CoA synthesis, lipid metabolism and lipotoxicity.
- Lipid signals and insulin resistance.
- Acyl-CoA metabolism and partitioning.
- Glycerol-3-phosphate acyltransferase-4-deficient mice are protected from diet-induced insulin resistance by the enhanced association of mTOR and rictor.
- Glycerol-3-phosphate Acyltransferase Isoform-4 (GPAT4) Limits Oxidation of Exogenous Fatty Acids in Brown Adipocytes.
- Physiological Consequences of Compartmentalized Acyl-CoA Metabolism.
- Long-chain acyl-CoA synthetase 2 knockdown leads to decreased fatty acid oxidation in fat body and reduced reproductive capacity in the insect Rhodnius prolixus.
- Deficiency of glycerol-3-phosphate acyltransferase 1 decreases triacylglycerol storage and induces fatty acid oxidation in insect fat body.
- Long-chain acyl-CoA synthetase isoforms differ in preferences for eicosanoid species and long-chain fatty acids.
- Arsenite and methylarsonite inhibit mitochondrial metabolism and glucose-stimulated insulin secretion in INS-1 832/13 β cells.
Fellowships
- Medical University of South Carolina, Endocrinology, Diabetes and Metabolism, SC, USA 2005
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