Dr. Andrew I. Schafer MD
Hematologist-Oncologist | Hematology & Oncology
525 E 68th St M522 New York NY, 10065About
Dr. Andrew Schafer is a hematologist oncologist practicing in New York, NY. Dr. Schafer specializes in the diagnosis, treatment and prevention of blood diseases such as anemia, hemophilia, sickle-cell disease, leukemia and lymphoma. Hematologist Oncologists are also trained in the study of cancer and its attack on other organs.
Education and Training
M.D. (?),University of Pennsylvania, MD 1973
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Hematology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Venous thrombosis as a chronic disease.
- Effects of nonsteroidal anti-inflammatory therapy on platelets.
- Platelet-derived growth factor stimulates heme oxygenase-1 gene expression and carbon monoxide production in vascular smooth muscle cells.
- Physiological cyclic stretch causes cell cycle arrest in cultured vascular smooth
- Physiological cyclic stretch directs L-arginine transport and metabolism to collagen synthesis in vascular smooth muscle.
- YC-1, a benzyl indazole derivative, stimulates vascular cGMP and inhibits neointima formation.
- Transforming growth factor-beta(1) stimulates L-arginine transport and metabolism in vascular smooth muscle cells: role in polyamine and collagen synthesis.
- Heme oxygenase-1 attenuates vascular remodeling following balloon injury in rat carotid arteries.
- Antithrombotic therapy in patients with acute coronary syndromes.
- Transforming growth factor-beta 1 stimulates vascular smooth muscle cell L-proline transport by inducing system A amino acid transporter 2 (SAT2) gene expression.
- Genetic polymorphisms in arterial thrombosis and vascular disease.
- Adenovirus-mediated heme oxygenase-1 gene delivery inhibits injury-induced vascular neointima formation.
- Thrombocytosis and thrombocythemia.
- Cyclin A transcriptional suppression is the major mechanism mediating homocysteine-induced endothelial cell growth inhibition.
- YC-1-mediated vascular protection through inhibition of smooth muscle cell proliferation and platelet function.
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