Dr. Katrin Ingrid Andreasson M.D.
Neurologist | Neurology
1800 Orleans St Baltimore MD, 21287About
Dr. Katrin Andreasson is a distinguished Neurologist in Baltimore, MD. Dr. Andreasson specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Andreasson employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Andreasson is committed to staying abreast of the latest developments in neurological research and therapies.
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Arcadlin is a neural activity-regulated cadherin involved in long term potentiation.
- Egr3/Pilot, a zinc finger transcription factor, is rapidly regulated by activity in brain neurons and colocalizes with Egr1/zif268.
- Age-dependent cognitive deficits and neuronal apoptosis in cyclooxygenase-2 transgenic mice.
- Role of immediate early gene expression in cortical morphogenesis and plasticity.
- Cyclooxygenase-dependent lipid-modification of brain proteins.
- Prostaglandin H(2)-derived adducts of proteins correlate with Alzheimer's disease severity.
- Cycloxygenase-2 activity promotes cognitive deficits but not increased amyloid burden in a model of Alzheimer's disease in a sex-dimorphic pattern.
- Intracellular trafficking and processing of pro-opiomelanocortin.
- Amyloid beta protein-induced zinc sequestration leads to synaptic loss via dysregulation of the ProSAP2/Shank3 scaffold.
- Suppression of inflammation with conditional deletion of the prostaglandin E2 EP2 receptor in macrophages and brain microglia.
- Suppression of Alzheimer-associated inflammation by microglial prostaglandin-E2 EP4 receptor signaling.
- Production of pro-opiomelanocortin (POMC) by a vaccinia virus transient expression system and in vitro processing of the expressed prohormone by POMC-converting enzyme.
- Prostaglandin signaling suppresses beneficial microglial function in Alzheimer's disease models.
- Microarray analysis of the in vivo response of microglia to Aβ peptides in mice with conditional deletion of the prostaglandin EP2 receptor.
- Microglial malfunction: the third rail in the development of Alzheimer's disease.
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