Dr. Amber D. Van laar M.D.
Neurologist | Neurology
3501 5TH AVE PITTSBURGH PA, 15213Rating
4/5
About
Dr. Amber Van laar is a distinguished Neurologist in PITTSBURGH, PA. Dr. Van laar specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Van laar employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Van laar is committed to staying abreast of the latest developments in neurological research and therapies.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Detection of apoptosis and changes in mitochondrial membrane potential with chloromethyl-X-rosamine.
- Neurotrophin-3 contributes to the initiation of behavioral sensitization to cocaine by activating the Ras/Mitogen-activated protein kinase signal transduction cascade.
- The monoamine neurons of the rat brain preferentially express a splice variant of alpha1B subunit of the N-type calcium channel.
- Repeated stimulation of L-type calcium channels in the rat ventral tegmental area mimics the initiation of behavioral sensitization to cocaine.
- The role of neurotrophic factors in psychostimulant-induced behavioral and neuronal plasticity.
- OLETF (Otsuka Long-Evans Tokushima Fatty) rats that lack the CCK 1 (A) receptor develop less behavioral sensitization to repeated cocaine treatment than wild type LETO (Long Evans Tokushima Otsuka) rats.
- Determination of atmospheric isomeric polycyclic arenes by thin-layer chromatography and fluorescence spectrophotometry.
- Cocaine administered into the medial prefrontal cortex reinstates cocaine-seeking behavior by increasing AMPA receptor-mediated glutamate transmission in the nucleus accumbens.
- Tyrosine kinase B and C receptors in the neostriatum and nucleus accumbens are co-localized in enkephalin-positive and enkephalin-negative neuronal profiles and their expression is influenced by cocaine.
- Inhibition of NMDA-induced striatal dopamine release and behavioral activation by
- Dopamine-, NMDA- and sigma-receptor antagonists exert differential effects on basal and amphetamine-induced changes in neostriatal ascorbate and DOPAC in awake, behaving rats.
- NOTE ON TESTING CONDITIONS.
- Unilateral neostriatal kainate, but not 6-OHDA, lesions block dopamine agonist-induced ascorbate release in the neostriatum of freely moving rats.
- Cocaine-induced alterations in dopamine receptor signaling: implications for reinforcement and reinstatement.
- D1-like and D2 dopamine receptor antagonists administered into the shell subregion of the rat nucleus accumbens decrease cocaine, but not food, reinforcement.
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