Daniel Jeremy Lerner M.D.
Cardiologist | Cardiovascular Disease
223 Madison Rd Scarsdale NY, 10583About
Dr. Daniel Lerner is a cardiologist practicing in Scarsdale, NY. Dr. Lerner specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Lerner also practices preventative medicine, helping patients maintain a heart-healthy life.
Education and Training
University of Miami Leonard M. Miller School of Medicine 1992
University of California 1990
Board Certification
RadiologyAmerican Board of RadiologyABR
Provider Details
Expert Publications
Data provided by the National Library of Medicine- RNA interference reveals that endogenous Xenopus MinK-related peptides govern mammalian K+ channel function in oocyte expression studies.
- The Rho guanine nucleotide exchange factor Lsc homo-oligomerizes and is negatively regulated through domains in its carboxyl terminus that are absent in novel splenic isoforms.
- MinK-related peptide 2 modulates Kv2.1 and Kv3.1 potassium channels in mammalian brain.
- Rho GEF Lsc is required for normal polarization, migration, and adhesion of formyl-peptide-stimulated neutrophils.
- The KCNE2 potassium channel ancillary subunit is essential for gastric acid secretion.
- Targeted deletion of kcne2 impairs ventricular repolarization via disruption of I(K,slow1) and I(to,f).
- MinK-dependent internalization of the IKs potassium channel.
- Kcne2 deletion uncovers its crucial role in thyroid hormone biosynthesis.
- Targeted deletion of Kcne2 causes gastritis cystica profunda and gastric neoplasia.
- Genetic dissection reveals unexpected influence of beta subunits on KCNQ1 K+ channel polarized trafficking in vivo.
- KCNE2 forms potassium channels with KCNA3 and KCNQ1 in the choroid plexus epithelium.
- Mortality and cost associated with cardiovascular implantable electronic device infections.
- Comparison of mortality in women versus men with infections involving cardiovascular implantable electronic device.
- KCNQ1, KCNE2, and Na+-coupled solute transporters form reciprocally regulating complexes that affect neuronal excitability.
- Increased long-term mortality in patients with cardiovascular implantable electronic device infections.
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