Dr. Bradford S Mcgwire M.D.
Infectious Disease Specialist | Infectious Disease
456 W 10th Ave Columbus OH, 43210About
Dr. Bradford Mcgwire is an infectious disease specialist practicing in Columbus, OH. Dr. Mcgwire specializes in infections that are difficult to diagnose or unresponsive to treatments, such as HIV or airborne infections from a foreign country. Infectious disease specialists usually work with conditions that are not treatable by a primary physician but it is important to keep contact with the primary physician in order to receive information about the patients history and for deciding which diagnostic tests are appropriate.
Education and Training
/,Grant Review. Evaluator. Aha-Grant Review. Dallas, Tx. MD
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Infectious Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Extracellular release of the glycosylphosphatidylinositol (GPI)-linked Leishmania surface metalloprotease, gp63, is independent of GPI phospholipolysis: implications for parasite virulence.
- Molecular determinants and regulation of Leishmania virulence.
- Migration through the extracellular matrix by the parasitic protozoan Leishmania is enhanced by surface metalloprotease gp63.
- Leishmania model for microbial virulence: the relevance of parasite multiplication and pathoantigenicity.
- Infliximab and reactivation of cerebral toxoplasmosis.
- Infection of polarized MDCK cells with herpes simplex virus 1: two asymmetrically distributed cell receptors interact with different viral proteins.
- The major surface-metalloprotease of the parasitic protozoan, Leishmania, protects against antimicrobial peptide-induced apoptotic killing.
- Fibronectin binding and proteolytic degradation by Leishmania and effects on macrophage activation.
- Trypanosoma cruzi GP63 proteins undergo stage-specific differential posttranslational modification and are important for host cell infection.
- Antimicrobial peptide-induced apoptotic death of leishmania results from calcium-de pend ent, caspase-independent mitochondrial toxicity.
- Interactions of antimicrobial peptides with Leishmania and trypanosomes and their functional role in host parasitism.
- Mammalian antimicrobial peptide influences control of cutaneous Leishmania infection.
- Modes of action of Leishmanicidal antimicrobial peptides.
- Secreted trypanosome cyclophilin inactivates lytic insect defense peptides and induces parasite calcineurin activation and infectivity.
- Voriconazole suppresses the growth of Leishmania species in vitro.
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