Dr. Gregory Fuller is a Neuropsychiatrist practicing in Houston, TX. Dr. Fuller studies, evaluates, diagnoses, and treats mental disorders attributable to diseases of the nervous system. Neuropsychiatrists are trained to treat disorders occurring in patients due to irritability, attention deficit disorder, epilepsy, and many other conditions.
Education and Training
Baylor College of Medicine 1987
PathologyAmerican Board of PathologyABP
- Insulin-like growth factor binding protein 2: gene expression microarrays and the hypothesis-generation paradigm.
- Tissue microarrays: applications in neuropathology research, diagnosis, and education.
- Constitutive activation of Stat3alpha in brain tumors: localization to tumor endothelial cells and activation by the endothelial tyrosine kinase receptor (VEGFR-2).
- Necrosis and glioblastoma: a friend or a foe? A review and a hypothesis.
- Toward better early-phase brain tumor clinical trials: a reappraisal of current methods and proposals for future strategies.
- Demyelinating pseudotumor.
- Identification of combination gene sets for glioma classification.
- High-throughput molecular profiling of high-grade astrocytomas: the utility of fluorescence in situ hybridization on tissue microarrays (TMA-FISH).
- Melanoma metastatic to central neurocytoma: a novel case of tumor-to-tumor metastasis.
- Insulin-like growth factor binding protein 2 enhances glioblastoma invasion by activating invasion-enhancing genes.
- Identification of necrosis-associated genes in glioblastoma by cDNA microarray analysis.
- Cystic glioblastoma multiforme: survival outcomes in 22 cases.
- Expression of the splicing regulator polypyrimidine tract-binding protein in normal and neoplastic brain.
- Primitive neuroectodermal tumors, embryonal tumors, and other small cell and poorly differentiated malignant neoplasms of the central and peripheral nervous systems.
- Differential gene and protein expression in primary breast malignancies and their lymph node metastases as revealed by combined cDNA microarray and tissue microarray analysis.
- Analysis of the activation status of Akt, NFkappaB, and Stat3 in human diffuse
- Textiloma (gossypiboma) mimicking recurrent intracranial tumor.
- Orbital and optic chiasmal extension of uveal melanoma.
- The mitosis-specific antibody anti-phosphohistone-H3 (PHH3) facilitates rapid reliable grading of meningiomas according to WHO 2000 criteria.
- Loss of the AP-2alpha transcription factor is associated with the grade of human gliomas.
- Elevated expression of p21 (WAF1/Cip1) in hormonally active pituitary adenomas.
- A mutation-created novel intra-exonic pre-mRNA splice site causes constitutive activation of KIT in human gastrointestinal stromal tumors.
- Inactivation of the invasion inhibitory gene IIp45 by alternative splicing in gliomas.
- Intracranial extension of acinic cell carcinoma of the parotid gland.
- Misdiagnosis of olfactory neuroblastoma.
- Attenuated expression of DFFB is a hallmark of oligodendrogliomas with 1p-allelic loss.
- Pineal cyst apoplexy: case report and review of the literature.
- The SHREW1 gene, frequently deleted in oligodendrogliomas, functions to inhibit cell adhesion and migration.
- An interaction between insulin-like growth factor-binding protein 2 (IGFBP2) and integrin alpha5 is essential for IGFBP2-induced cell mobility.
- Survival prediction in patients with glioblastoma multiforme by human telomerase genetic variation.
- Origin of chordoid glioma of the third ventricle.
- Pulmonary artery sarcoma: a clinicopathologic and immunohistochemical study of 12 cases.
- Pathway alterations during glioma progression revealed by reverse phase protein lysate arrays.
- Randomized, double-blind, placebo-controlled trial of marimastat in glioblastoma multiforme patients following surgery and irradiation.
- Overexpression of IGFBP5, but not IGFBP3, correlates with the histologic grade of human diffuse glioma: a tissue microarray and immunohistochemical study.
- Insulin-like growth factor-binding protein 2 and 5 are differentially regulated in ovarian cancer of different histologic types.
- Sustained angiopoietin-2 expression disrupts vessel formation and inhibits glioma growth.
- Narrowing the differential diagnosis in surgical neuropathology.
- Transgenic E2F1 expression in the mouse brain induces a human-like bimodal pattern of tumors.
- The 2007 Revised World Health Organization (WHO) Classification of Tumours of the Central Nervous System: newly codified entities.
- Newly codified glial neoplasms of the 2007 WHO Classification of Tumours of the Central Nervous System: angiocentric glioma, pilomyxoid astrocytoma and pituicytoma.
- Insulin-like growth factor binding protein 2 promotes glioma development and progression.
- Shrinkage of germinoma by dexamethasone only.
- Constitutive activation of c-Jun N-terminal kinase correlates with histologic grade and EGFR expression in diffuse gliomas.
- Knockdown of GluR1 expression by RNA interference inhibits glioma proliferation.
- Medullary thyroid carcinoma metastatic to the pituitary gland: an unusual site of metastasis.
- The WHO Classification of Tumours of the Central Nervous System, 4th edition.
- Intracranial Rosai-Dorfman disease with relapsing spinal lesions.
- Incidence and prognostic impact of FoxP3+ regulatory T cells in human gliomas.
- Carcinoid tumors arising in tailgut cysts may be associated with estrogen receptor status: case report and review of the literature.
- Wernicke's encephalopathy: an underrecognized and reversible cause of confusional state in cancer patients.
- Surgical management of lateral-ventricle metastases: report of 29 cases in a single-institution experience.
- IGFBP2 is a candidate biomarker for Ink4a-Arf status and a therapeutic target for high-grade gliomas.
- Immunohistochemical detection of somatostatin receptor subtype 5 (SSTR-5) in cushing adenoma.
- Spontaneous canine gliomas: overexpression of EGFR, PDGFRalpha and IGFBP2 demonstrated by tissue microarray immunophenotyping.
- IIp45 inhibits cell migration through inhibition of HDAC6.
- The role of tregs in human glioma patients and their inhibition with a novel STAT-3 inhibitor.
- JNK mediates insulin-like growth factor binding protein 2/integrin alpha5-dependent glioma cell migration.
- Myoblast-derived neuronal cells form glutamatergic neurons in the mouse cerebellum.
- Endolymphatic sac tumor (aggressive papillary tumor of middle ear and temporal bone): sine qua non radiology-pathology and the University of Texas MD Anderson Cancer Center experience.
- Systemic combinatorial peptide selection yields a non-canonical iron-mimicry mechanism for targeting tumors in a mouse model of human glioblastoma.
- Oncogene interactions are required for glioma development and progression as revealed by a tissue specific transgenic mouse model.
- Tumor-associated methylation of the putative tumor suppressor AJAP1 gene and association between decreased AJAP1 expression and shorter survival in patients with glioma.
- A GATA4-regulated tumor suppressor network represses formation of malignant human astrocytomas.
- The high mitotic count detected by phospho-histone H3 immunostain does not alter the benign behavior of angiocentric glioma.
- Absence of IDH1-R132H mutation predicts rapid progression of nonenhancing diffuse glioma in older adults.
- Insulin-like growth factor-binding protein 2-driven glioma progression is prevented by blocking a clinically significant integrin, integrin-linked kinase, and NF-κB network.
- Isocitrate dehydrogenase 1 R132H mutation is not detected in angiocentric glioma.
- Giant cell ependymoma-report of three cases and review of the literature.
- Curvularia brain abscess.
- Transcriptome and small RNA deep sequencing reveals deregulation of miRNA biogenesis in human glioma.
- Brain metastases from papillary thyroid carcinomas.
- Clinical and prognostic features of adult patients with gangliogliomas.
- Survivin transcript variant 2 drives angiogenesis and malignant progression in proneural gliomas.
- Anti-vascular endothelial growth factor therapy-induced glioma invasion is associated with accumulation of Tie2-expressing monocytes.
- Synchronous rosette-forming glioneuronal tumor and diffuse astrocytoma with molecular characterization: a case report.
- International Society Of Neuropathology--Haarlem consensus guidelines for nervous system tumor classification and grading.
- Genomically amplified Akt3 activates DNA repair pathway and promotes glioma progression.
- Primary rhabdomyosarcoma of the pineal gland.
- Genetic, epigenetic, and molecular landscapes of multifocal and multicentric glioblastoma.
- Gliomatosis peritonei: a clinicopathologic and immunohistochemical study of 21 cases.
- Primary Glial and Neuronal Tumors of the Ovary or Peritoneum: A Clinicopathologic Study of 11 Cases.
- Driver or passenger effects of augmented c-Myc and Cdc20 in gliomagenesis.
- Analysis of the inhibitors of apoptosis identifies BIRC3 as a facilitator of malignant progression in glioma.
- Glioma progression is mediated by an addiction to aberrant IGFBP2 expression and can be blocked using anti-IGFBP2 strategies.
- Fulminant spinal cord compression caused by postradiation inflammatory pseudotumor with rapid response to steroids: case report.
- Whole-transcriptome analysis of chordoma of the skull base.
- Sarcoma Brain Metastases: 28 Years of Experience at a Single Institution.
- Epigenetic Activation of WNT5A Drives Glioblastoma Stem Cell Differentiation and Invasive Growth.
- Isolated Ectopic Cutaneous Atypical Meningioma of the Scalp: Another Mimicker of Primary Adnexal Tumor.
- Identification of Histological Correlates of Overall Survival in Lower Grade Gliomas Using a Bag-of-words Paradigm: A Preliminary Analysis Based on Hematoxylin & Eosin Stained Slides from the Lower Grade Glioma Cohort of The Cancer Genome Atlas.
- Molecular Classification of Adult Diffuse Gliomas: Conflicting IDH1/IDH2, ATRX and 1p/19q Results.
- Transcriptome comparison identifies potential biomarkers of spine and skull base chordomas.
- Multimodality Management of Recurrent Skull Base Chordomas: Factors Impacting Tumor Control and Disease-Specific Survival.
- Extending the Neuroanatomic Territory of Diffuse Midline Glioma, K27M Mutant: Pineal Region Origin.
- Duke Univ Med Ctr, Anatomic And Clinical Pathology
- Duke University Med Center
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- Causes and Treatment for Gynecomastia
What Is Gynecomastia?When boys enter adolescence, they will see a lot of hormonal changes in their body. The growth in both maternal and paternal hormones leads to changes like a growth of hair, deepening of the voice, etc. In some cases, due to excess estrogen in the body, the breast tissue in...
- Lyrica for Neuropathy
What is Lyrica?Lyrica, also known as pregabalin, is a drug made for the treatment of seizures in epilepsy. This drug is made to work by restricting nerve impulses that induce seizures. It can also inhibit pain by suppressing pain signal transmissions by the nerves....
- Research Suggests Broccoli Could Benefit Crohn's Disease
There is a common myth that an individual’s diet can cause Crohn’s disease. This rumor is popular because on the surface it seems to make sense: Diet is often the culprit when it comes to stomach-related illness. However, medical research has proven that a person’s diet will not cause them to...
- What Is Normal Pressure Hydrocephalus?
Normal pressure hydrocephalus (NPH) is a type of brain disorder, wherein excess cerebrospinal fluid builds up in the ventricles of the brain. It causes problems in reasoning, thinking, difficulty in walking, and loss of bladder control.Brains ventricles are hollow fluid-filled chambers. Even...
- What is a Transient Ischemic Attack?
Also considered as a mini stroke, a transient ischemic attack (TIA) has symptoms similar to that of a stroke, but lasting only for a short period of time, without causing any permanent damage. TIA is often caused by the interruption of blood flow to the brain by a blood clot. Once the blood flow is...
- Tips On Living With Epilepsy
Get to Know Your EpilepsyEpilepsy can affect people at any point in their life. There’s no specific race or gender that have a higher incidence rate of epilepsy. Until now, there was no single cause of epilepsy. The causes of epilepsy range from head injuries to birth defects to tumors and...