Dr. Joel M Weinberg MD
Nephrologist (Kidney Specialist) | Nephrology
1500 East Medical Center Dr 3rd Floor Taubman Ct Ann Arbor MI, 48109Rating
1/5
About
Dr. Joel Weinberg is a nephrologist practicing in Ann Arbor, MI. Dr. Weinberg specializes in the care and treatment of the kidneys. As a nephrologist, Dr. Weinberg most typically treats conditions like kidney stones, chronic kidney disease, acute renal failure, polycystuc kidney disease, high blood pressure and more. Nephrologists are also experts on kidney transplantation and dialysis. They are usually referred to by primary care physicians for problems related to the kidneys, and while they can perform tests to diagnose kidney disorders, they do not perform surgeries.
Education and Training
Harvard Med Sch, Boston Ma 1976
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Pulmonary Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Site-specific alteration of actin assembly visualized in living renal epithelial cells during ATP depletion.
- Glycine protection of PC-12 cells against injury by ATP-depletion.
- Recent advances in the pathophysiology of ischemic acute renal failure.
- Mitochondrial function.
- Preservation of complex I function during hypoxia-reoxygenation-induced mitochondrial injury in proximal tubules.
- Assessment of mitochondrial membrane potential in proximal tubules after hypoxia-reoxygenation.
- F1FO-ATPase activity and ATP dependence of mitochondrial energization in proximal tubules after hypoxia/reoxygenation.
- Accumulation of nonesterified fatty acids causes the sustained energetic deficit in kidney proximal tubules after hypoxia-reoxygenation.
- A role for AMP-activated protein kinase in diabetes-induced renal hypertrophy.
- Alleviation of fatty acid and hypoxia-reoxygenation-induced proximal tubule deenergization by ADP/ATP carrier inhibition and glutamate.
- Calcium in cell injury and death.
- Evidence for involvement of nonesterified fatty acid-induced protonophoric uncoupling during mitochondrial dysfunction caused by hypoxia and reoxygenation.
- Mitochondrial dysfunction induced by pancreatic and crotalic (Crotalus durissus terrificus) phospholipases A2 on rabbit proximal tubules suspensions.
- Inhibition of autoregulated TGFbeta signaling simultaneously enhances proliferation and differentiation of kidney epithelium and promotes repair following renal ischemia.
- Regulation of the mitochondrial permeability transition in kidney proximal
Dr. Joel M Weinberg MD's Practice location
Practice At 1500 East Medical Center Dr 3rd Floor Taubman Ctr Recp C
1500 East Medical Center Dr 3rd Floor Taubman Ct -Ann Arbor, MI 48109Get Direction
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