Kevin G Brown
Counselor/Therapist | Mental Health
81840 AVENUE 46 INDIO CA, 92201About
Kevin Brown is an Addiction Medicine Physician in INDIO, CA. Kevin evaluates patients using many different procedures, in order to determine what treatments must be carried out in order to properly assess their symptoms. Addiction Medicine Physicians provide medical care in addition to consultation for each patient and their families.
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Expert Publications
Data provided by the National Library of Medicine- Catholic charities.
- Menkes Copper ATPase (Atp7a) is a novel metal-responsive gene in rat duodenum, and immunoreactive protein is present on brush-border and basolateral membrane domains.
- Development of a chronic inhalation reference level for respirable crystalline silica.
- Gene chip analyses reveal differential genetic responses to iron deficiency in rat duodenum and jejunum.
- Mist delivery of eye medication to the anterior segment.
- Promoter analysis of intestinal genes induced during iron-deprivation reveals enrichment of conserved SP1-like binding sites.
- Prediction of synergistic transcription factors by function conservation.
- Novel insights into intestinal and renal folate transport. Focus on "Apical membrane targeting and trafficking of the human proton-coupled folate transporter in polarized epithelia".
- Induction of arachidonate 12-lipoxygenase (Alox15) in intestine of iron-deficient rats correlates with the production of biologically active lipid mediators.
- Hepcidin regulation of iron transport.
- Alternative splicing of the Menkes copper Atpase (Atp7a) transcript in the rat intestinal epithelium.
- Metabolic crossroads of iron and copper.
- Cross-species comparison of genomewide gene expression profiles reveals induction of hypoxia-inducible factor-responsive genes in iron-deprived intestinal epithelial cells.
- Transcriptional regulation of the Menkes copper ATPase (Atp7a) gene by hypoxia-inducible factor (HIF2{alpha}) in intestinal epithelial cells.
- Serum ceruloplasmin protein expression and activity increases in iron-deficient rats and is further enhanced by higher dietary copper intake.
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