Robert Pollmann PA-C
Physician Assistant
600 Robbins Rd Boise ID, 83702About
Robert Pollmann is a physician assistant practicing in BOISE, ID. Robert specializes in preventing and treating human illness and injury by providing a broad range of care under the supervision of a physician. A physician assistant's work can include physical exams, ordering and interpreting tests, performing procedures and prescribing medication. Often times, they can assist in surgery but the scope of practice can vary according to jurisdiction or health care setting.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Vasopressin action in the mammalian cerebral cortex.
- In vivo and in vitro hyperbaric studies in mice suggest novel sites of action for ethanol.
- A women's health issue: Alzheimer's disease and strategies for maintaining cognitive health.
- The estrogen replacement therapy of the Women's Health Initiative promotes the cellular mechanisms of memory and neuronal survival in neurons vulnerable to Alzheimer's disease.
- Vasopressin and oxytocin receptor mRNA expression during rat telencephalon development.
- Spatial, cellular and temporal basis of vasopressin potentiation of norepinephrine-induced cAMP formation.
- Vasopressin-induced neurotrophism in cultured neurons of the cerebral cortex: dependency on calcium signaling and protein kinase C activity.
- Cellular and molecular mechanisms of estrogen regulation of memory function and neuroprotection against Alzheimer's disease: recent insights and remaining challenges.
- Regulation and mechanism of L-type calcium channel activation via V1a vasopressin receptor activation in cultured cortical neurons.
- 17beta-estradiol induces Ca2+ influx, dendritic and nuclear Ca2+ rise and subsequent cyclic AMP response element-binding protein activation in hippocampal neurons: a potential initiation mechanism for estrogen neurotrophism.
- 17Beta-estradiol induced Ca2+ influx via L-type calcium channels activates the Src/ERK/cyclic-AMP response element binding protein signal pathway and BCL-2 expression in rat hippocampal neurons: a potential initiation mechanism for estrogen-induced n
- 17beta-Estradiol potentiates field excitatory postsynaptic potentials within each subfield of the hippocampus with greatest potentiation of the associational/commissural afferents of CA3.
- 17beta-Estradiol Induction of Filopodial Growth in Cultured Hippocampal Neurons within Minutes of Exposure.
- Selective oestrogen receptor modulators differentially potentiate brain mitochondrial function.
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