Dr. Marc E Goldyne M.D., PH.D.
Doctor
2299 Post St Suite 310 San Francisco CA, 94115About
Dr. Marc E Goldyne M.D., PH.D. is a top Doctor in San Francisco, CA. With a passion for the field and an unwavering commitment to their specialty, Dr. Marc E Goldyne M.D., PH.D. is an expert in changing the lives of their patients for the better. Through their designated cause and expertise in the field, Dr. Marc E Goldyne M.D., PH.D. is a prime example of a true leader in health care. As a leader and expert in their field, Dr. Marc E Goldyne M.D., PH.D. is passionate about enhancing patient quality of life. They embody the values of communication, safety, and trust when dealing directly with patients. In San Francisco, CA, Dr. Marc E Goldyne M.D., PH.D. is a true asset to their field and dedicated to the profession of medicine.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Cyclooxygenase isoforms in human skin.
- IgE in human eccrine sweat.
- Evaluation of an asynchronous teleconsultation system for diagnosis of skin cancer and other skin diseases.
- Prostaglandins and chemotaxis: enhancement of polymorphonuclear leukocyte chemotaxis by prostaglandin F2alpha.
- Peroxisomal abnormality in fibroblasts from involved skin of CHILD syndrome. Case study and review of peroxisomal disorders in relation to skin disease.
- Norepinephrine-induced increase in sympathetic neuron-derived prostaglandins is independent of neuronal release mechanisms.
- Noradrenaline-induced prostaglandin production by sympathetic postganglionic neurons is mediated by alpha 2-adrenergic receptors.
- Prostaglandins and cutaneous inflammation.
- Prostaglandins and cutaneous inflammation.
- Keratinocytes can regulate prostaglandin synthesis by fibroblasts: potential role for interleukin 1.
- CHILD syndrome: fibroblasts from involved skin fail to proliferate in response to interleukin 1a due to induction of excessive prostaglandin E2 synthesis.
- Dermatology: leukotrienes-their importance in dermatology.
- Noxious stimulus-induced increase in spinal prostaglandin E2 is noradrenergic terminal-dependent.
- Prostaglandin E modulation of the mitogenic response of human T cells. Differential response of T-cell subpopulations.
- Prostaglandin E modulation of the mitogenic response of human T cells. Differential response of T-cell subpopulations.
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