Dr. Michael E Greenberg MD
Emergency Physician | Medical Toxicology
230 N Broad St Drexel Emergency Med Philadelphia PA, 19102About
Dr. Michael Greenberg practices Emergency Medicine in Philadelphia, PA. Dr. Greenberg assesses patients who seek immediate medical attention at any time of day or night. Emergency Medicine Physicians are trained to efficiently work with each patient and situation no matter how acute or life-threatening. Dr. Greenberg examines patients, determines means of testing, diagnoses conditions, and decides the best treatment methods.
Education and Training
Temple Univ Sch of Med, Philadelphia Pa 1976
Board Certification
Emergency MedicineAmerican Board of Emergency MedicineABEM
Preventative MedicineAmerican Board of Preventative MedicineABPM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Modulation of NMDA receptor-dependent calcium influx and gene expression through EphB receptors.
- A calcium-responsive transcription factor, CaRF, that regulates neuronal activity-dependent expression of BDNF.
- DNA repair pathway stimulated by the forkhead transcription factor FOXO3a through the Gadd45 protein.
- CREB transcriptional activity in neurons is regulated by multiple, calcium-specific phosphorylation events.
- The IGF-1/Akt pathway is neuroprotective in Huntington's disease and involves Huntingtin phosphorylation by Akt.
- Analysis of phosphorylation-dependent protein-protein interactions using a bacterial two-hybrid system.
- Time-lapse imaging of a dynamic phosphorylation-dependent protein-protein interaction in mammalian cells.
- Survival factor-mediated BAD phosphorylation raises the mitochondrial threshold for apoptosis.
- Regulation of transcription factors by neuronal activity.
- The many forks in FOXO's road.
- Upstream stimulatory factors are mediators of Ca2+-responsive transcription in neurons.
- Basic helix-loop-helix factors in cortical development.
- Bad-deficient mice develop diffuse large B cell lymphoma.
- The Drosophila forkhead transcription factor FOXO mediates the reduction in cell number associated with reduced insulin signaling.
- BAD and glucokinase reside in a mitochondrial complex that integrates glycolysis and apoptosis.
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