Barry E. Kosofsky MPH, PHD
Neurologist | Neurology with Special Qualifications in Child Neurology
428 E 72nd St New York NY, 10021Rating
3/5
About
Dr. Barry Kosofsky is a distinguished Neurologist in New York, NY. Dr. Kosofsky specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Kosofsky employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Kosofsky is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
Johns Hopkins University School of Medicine 1985
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Mechanisms of action of drugs of abuse on the developing fetal brain.
- Cocaine activation discriminates dopaminergic projections by temporal response: an fMRI study in Rat.
- Developmental considerations of neurotoxic exposures.
- Chronic fluoxetine treatment selectively uncouples raphe 5-HT(1A) receptors as measured by [(35)S]-GTP gamma S autoradiography.
- Increased vulnerability to self-administer cocaine in mice prenatally exposed to cocaine.
- Ventilatory control in newborn mice prenatally exposed to cocaine.
- Cocaine-induced disturbances of corticogenesis in the developing murine brain.
- Does drug abuse beget drug abuse? Behavioral analysis of addiction liability in animal models of prenatal drug exposure.
- Altered cocaine-induced behavioral sensitization in adult mice exposed to cocaine in utero.
- Cocaine exposure decreases GABA neuron migration from the ganglionic eminence to the cerebral cortex in embryonic mice.
- The effect of prenatal cocaine exposure on the stress response of adult mice.
- BDNF-induced white matter neuroprotection and stage-dependent neuronal survival following a neonatal excitotoxic challenge.
- L-type Ca2+ channels mediate adaptation of extracellular signal-regulated kinase 1/2 phosphorylation in the ventral tegmental area after chronic amphetamine treatment.
- Neuropathological consequences of prenatal cocaine exposure in the mouse.
- Augmented constitutive CREB expression in the nucleus accumbens and striatum may contribute to the altered behavioral response to cocaine of adult mice exposed to cocaine in utero.
Treatments
- Headaches
- Epilepsy
- Learning Disabilities
- Concussion
- Birth Defects
- Migraine
- Lumecca
Barry E. Kosofsky MPH, PHD's Practice location
Practice At 428 E 72nd St
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