Dr. Jeffery Todd Harmon DMD
Dentist
255 W 36th St Jasper IN, 47546About
Dr. Jeffery Harmon is a Dentist practicing in Jasper, IN. Dr. Harmon specializes in preventing, diagnosing, and treating diseases and conditions associated with the mouth and overall dental health. Dentists are trained to carry out such treatment as professional cleaning, restorative, prosthodontic, and endodontic procedures, and performing examinations, among many others.
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Expert Publications
Data provided by the National Library of Medicine- Effect of insulin to decrease glucose transport in dissociated cells from the R3230AC mammary adenocarcinoma of diabetic rats.
- Effect of insulin to decrease glucose transport in dissociated cells from the R3230AC mammary adenocarcinoma of diabetic rats.
- Identification and characterization of the insulin receptor in the R3230AC mammary adenocarcinoma of the rat.
- Identification and characterization of the insulin receptor in the R3230AC mammary adenocarcinoma of the rat.
- Isolation of human platelet plasma membranes by glycerol lysis.
- Insulin binding and glucose transport in the R3230AC mammary adenocarcinoma.
- Insulin binding and glucose transport in the R3230AC mammary adenocarcinoma.
- Radiation inactivation experiments predict that a large aggregate form of the insulin receptor is a highly active tyrosine-specific protein kinase.
- Platelet activation by thrombin in the absence of the high-affinity thrombin receptor.
- Thrombin binds to a high-affinity approximately 900 000-dalton site on human platelets.
- Activation of platelets by alpha-thrombin is a receptor-mediated event. D-phenylalanyl-L-prolyl-L-arginine chloromethyl ketone-thrombin, but not N alpha-tosyl-L-lysine chloromethyl ketone-thrombin, binds to the high affinity thrombin receptor.
- Platelet activation by alpha-thrombin is a receptor-mediated event.
- A monomer-dimer model explains the results of radiation inactivation: binding characteristics of insulin receptor purified from human placenta.
- Identification of high-affinity (Kd 0.35 mumol/L) and low-affinity (Kd 7.9 mumol/L) platelet binding sites for ADP and competition by ADP analogues.
- Thrombin binding and response in platelets from patients with dyslipoproteinemias: increased stimulus-response coupling in type II hyperlipoproteinemia.
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