Dr. Michael Halassa is a psychiatrist practicing in Boston, MA. Dr. Halassa is a medical doctor specializing in the care of mental health patients. As a psychiatrist, Dr. Halassa diagnoses and treats mental illnesses. Dr. Halassa may treat patients through a variety of methods including medications, psychotherapy or talk therapy, psychosocial interventions and more, depending on each individual case. Different medications that a psychiatrist might prescribe include antidepressants, antipsychotic mediations, mood stabilizers, stimulants, sedatives and hypnotics. Dr. Halassa treats conditions like depression, anxiety, OCD, eating disorders, bipolar disorders, personality disorders, insomnia, ADD and other mental illnesses.
- The tripartite synapse: roles for gliotransmission in health and disease.
- Synaptic islands defined by the territory of a single astrocyte.
- Astrocytic modulation of sleep homeostasis and cognitive consequences of sleep loss.
- Tripartite synapses: roles for astrocytic purines in the control of synaptic physiology and behavior.
- Endogenous nonneuronal modulators of synaptic transmission control cortical slow
- Integrated brain circuits: astrocytic networks modulate neuronal activity and behavior.
- Integrated brain circuits: neuron-astrocyte interaction in sleep-related rhythmogenesis.
- Thalamocortical dynamics of sleep: roles of purinergic neuromodulation.
- Astrocyte-derived adenosine and A1 receptor activity contribute to sleep loss-induced deficits in hippocampal synaptic plasticity and memory in mice.
- Selective optical drive of thalamic reticular nucleus generates thalamic bursts and cortical spindles.
- Astrocyte regulation of sleep circuits: experimental and modeling perspectives.
- Chronic sleep restriction disrupts sleep homeostasis and behavioral sensitivity to alcohol by reducing the extracellular accumulation of adenosine.
- Design and fabrication of ultralight weight, adjustable multi-electrode probes for electrophysiological recordings in mice.
- The impact of NMDA receptor hypofunction on GABAergic neurons in the pathophysiology of schizophrenia.
- Response to: NMDA hypofunction attenuates driver inputs in higher order thalamic nuclei: An alternative view.
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