Dr. Stephen Albert Huang M.D.
Endocronologist (Pediatric) | Pediatric Endocrinology
300 Longwood Avenue Children's Hospital Boston MA, 02115Rating
5/5
About
Dr. Stephen Huang is a pediatric endocrinologist practicing in Boston, MA. Dr. Huang specializes in growth, puberty, diabetes or other disorders related to hormones that produce certain conditions in children and growing young adults. Pediatric endocrinologists possess copious knowledge on hormone chemicals and how they can affect other parts of the body and their functions.
Education and Training
Perelman School of Medicine at the University of Pennsylvania 1995
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Children with differentiated thyroid cancer achieve adequate hyperthyrotropinemia within 14 days of levothyroxine withdrawal.
- Type 2 iodothyronine deiodinase is the major source of plasma T3 in euthyroid humans.
- Physiology and pathophysiology of type 3 deiodinase in humans.
- Consumptive hypothyroidism caused by paraneoplastic production of type 3 iodothyronine deiodinase.
- The small polyphenolic molecule kaempferol increases cellular energy expenditure and thyroid hormone activation.
- Thyroid function disturbance and type 3 iodothyronine deiodinase induction after myocardial infarction in rats a time course study.
- Reawakened interest in type III iodothyronine deiodinase in critical illness and injury.
- Hypoxia-inducible factor induces local thyroid hormone inactivation during hypoxic-ischemic disease in rats.
- The thyroid hormone-inactivating deiodinase functions as a homodimer.
- Deiodinase-mediated thyroid hormone inactivation minimizes thyroid hormone signaling in the early development of fetal skeleton.
- Cellular and molecular basis of deiodinase-regulated thyroid hormone signaling.
- Can hyperthyroidism relapse after antithyroid drug treatment be predicted in children with Graves disease?
- Deiodination and cellular proliferation: parallels between development, differentiation, tumorigenesis, and now regeneration.
- Knockdown of the type 3 iodothyronine deiodinase (D3) interacting protein peroxiredoxin 3 decreases D3-mediated deiodination in intact cells.
- Soy protein diet alters expression of hepatic genes regulating fatty acid and thyroid hormone metabolism in the male rat.
Treatments
- Hashimoto's Thyroiditis
- Hyperthyroidism
- Thyroiditis
- Graves' Disease
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Nearest Hospitals
BRIGHAM AND WOMEN'S HOSPITALl
75 FRANCIS STREET BOSTON MA 2115